Dr. Richard Thomas Davey M.D.
Infectious Disease Specialist | Infectious Disease
10 Center Drive Msc 1460 Crc 4 1479 Bethesda MD, 20892About
Dr. Richard Davey is an infectious disease specialist practicing in Bethesda, MD. Dr. Davey specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Infectious Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Atovaquone suspension in HIV-infected volunteers: pharmacokinetics, pharmacodynamics, and TMP-SMX interaction study.
- Resistance to replication of human immunodeficiency virus challenge in SCID-Hu mice engrafted with peripheral blood mononuclear cells of nonprogressors is mediated by CD8(+) T cells and associated with a proliferative response to p24 antigen.
- Interleukin-2 up-regulates expression of the human immunodeficiency virus fusion
- Rapid activation of lymph nodes and mononuclear cell HIV expression upon interrupting highly active antiretroviral therapy in patients after prolonged viral suppression.
- Metabolic and anthropometric consequences of interruption of highly active antiretroviral therapy.
- Herpes zoster in an HIV-negative man on ritonavir.
- Impact of HIV-1 infection and highly active antiretroviral therapy on the kinetics of CD4+ and CD8+ T cell turnover in HIV-infected patients.
- A randomized, controlled 24-week study of intermittent subcutaneous interleukin-2 in HIV-1 infected patients in Thailand.
- Human immunodeficiency virus type 1 quasi species that rebound after discontinuation of highly active antiretroviral therapy are similar to the viral quasi species present before initiation of therapy.
- Acute hepatitis caused by a novel strain of hepatitis E virus most closely related to United States strains.
- CD4 T cell expansions are associated with increased apoptosis rates of T lymphocytes during IL-2 cycles in HIV infected patients.
- Evidence for increased T cell turnover and decreased thymic output in HIV infection.
- High-level HIV-1 viremia suppresses viral antigen-specific CD4(+) T cell proliferation.
- Diminished proliferation of human immunodeficiency virus-specific CD4+ T cells is associated with diminished interleukin-2 (IL-2) production and is recovered by exogenous IL-2.
- Phase I/II studies of the toxicity and immunogenicity of recombinant gp160 and p24 vaccines in HIV-infected individuals.
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