David E Barton MD
Internist
EMILE @ 42ND ST OMAHA NE, 68198About
Dr. David Barton is an internist practicing in OMAHA, NE. Dr. Barton specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Barton provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Intrafamilial phenotypic variability in Friedreich ataxia associated with a G130V mutation in the FRDA gene.
- Comparison of the CFTR mutation spectrum in three cohorts of patients of Celtic
- Detection of five common CFTR mutations by rapid-cycle real-time amplification refractory mutation system PCR.
- Uroplakin III is not a major candidate gene for primary vesicoureteral reflux.
- Best practice guidelines for the molecular genetic diagnosis of Type 1 (HFE-related) hereditary haemochromatosis.
- Emerging technologies for point-of-care genetic testing.
- Evaluation and use of a synthetic quality control material, included in the European external quality assessment scheme for cystic fibrosis.
- The increased incidence of the RET p.Gly691Ser variant in French-Canadian vesicoureteric reflux patients is not replicated by a larger study in Ireland.
- Preparation and validation of the first WHO international genetic reference panel for Fragile X syndrome.
- Multiallelic synthetic quality control material: lessons learned from the cystic fibrosis external quality assessment scheme.
- Establishment of the first WHO international genetic reference panel for Prader Willi and Angelman syndromes.
- In search of an uncultured human-associated TM7 bacterium in the environment.
- Genetics of vesicoureteral reflux.
- Quality assurance practices in Europe: a survey of molecular genetic testing laboratories.
- Clinical correlation and molecular evaluation confirm that the MLH1 p.Arg182Gly (c.544A>G) mutation is pathogenic and causes Lynch syndrome.
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