Prof. Jeremy Stuart Duffield M.D. PH.D.
Nephrologist (Kidney Specialist) | Nephrology
75 Francis St Brigham Medical Spec Boston MA, 02115About
Dr. Duffield is a UW Associate Professor of Medicine, Division of Nephrology Education InstitutionCredentialYear John Radcliffe Hospital (England)Medical education1992 Brigham and Women's HospitalFellowship ...
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Expert Publications
Data provided by the National Library of Medicine- Activated macrophages direct apoptosis and suppress mitosis of mesangial cells.
- The inflammatory macrophage: a story of Jekyll and Hyde.
- Low-dose cyclosporin therapy for recombinant erythropoietin-induced pure red-cell aplasia.
- Quantitative morphometry of lupus nephritis: the significance of collagen, tubular space, and inflammatory infiltrate.
- Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair.
- Mutations in complement factor I predispose to development of atypical hemolytic
- Scar-associated macrophages are a major source of hepatic matrix
- Endothelial activation markers in anemic non-dialysis chronic kidney disease patients.
- Pericytes and perivascular fibroblasts are the primary source of collagen-producing cells in obstructive fibrosis of the kidney.
- Bone marrow Ly6Chigh monocytes are selectively recruited to injured kidney and differentiate into functionally distinct populations.
- Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis.
- Macrophage Wnt7b is critical for kidney repair and regeneration.
- Serum amyloid P inhibits fibrosis through Fc gamma R-dependent monocyte-macrophage regulation in vivo.
- Mobilized human hematopoietic stem/progenitor cells promote kidney repair after ischemia/reperfusion injury.
- PRM-151 (recombinant human serum amyloid P/pentraxin 2) for the treatment of fibrosis.
Fellowships
- Peter Bent Brigham Hospital/Harvard University
- Royal Infirmary of Edinburgh
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