Dr. Dina J Zand MD
Geneticist | Clinical Genetics (M.D.)
111 Michigan Ave NW Washington DC, 20010Rating
3/5
About
Dr. Dina Zand practices Genetic Medicine in Washington, DC. As a geneticist, Dr. Zand performs experiments and analyzes data to interpret the inheritance of different traits in patients. A geneticist evaluates, diagnoses, and manages patients with hereditary conditions or congenital malformations, genetic risk calculations, and mutation analysis. Dr. Zand carries out studies, tests, and counsels patients with genetic diseases.
Board Certification
Medical GeneticsAmerican Board of Medical GeneticsABMG
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Crane-Heise syndrome: a second familial case report with elaboration of phenotype.
- Single-gene autosomal recessive disorders and Prader-Willi syndrome: an update for food and nutrition professionals.
- Complex management of a patient with a contiguous Xp11.4 gene deletion involving ornithine transcarbamylase: a role for detailed molecular analysis in complex presentations of classical diseases.
- Effectiveness of a clinical pathway for the emergency treatment of patients with inborn errors of metabolism.
- Automated Down syndrome detection using facial photographs.
- Hierarchical constrained local model using ICA and its application to Down syndrome detection.
- Digital facial dysmorphology for genetic screening: Hierarchical constrained local model using ICA.
- Severe neonatal holocarboxylase synthetase deficiency in west african siblings.
- Concurrent diagnoses of Prader-Willi syndrome and GM2 gangliosidosis caused by uniparental disomy of chromosome 15.
- An uncommon clinical presentation of relapsing dilated cardiomyopathy with identification of sequence variations in MYNPC3, KCNH2 and mitochondrial tRNA cysteine.
- Thiamine pyrophosphokinase deficiency causes a Leigh Disease like phenotype in a sibling pair: identification through whole exome sequencing and management strategies.
- Concurrent non-ketotic hyperglycinemia and propionic acidemia in an eight year old boy.
- T cell activation and increases in protein kinase C activity enhance retinoic acid-induced gene transcription.
- Dominant negative retinoid X receptor beta inhibits retinoic acid-responsive gene regulation in embryonal carcinoma cells.
- Retinoic acid-mediated down-regulation of Oct3/4 coincides with the loss of promoter occupancy in vivo.
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Practice At 111 Michigan Ave NW
111 Michigan Ave NW -Washington, DC 20010Get Direction
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