Dr. Tarun Ballubhai Patel DDS
Dentist
13279 Van Nuys Blvd Pacoima CA, 91331About
Dr. Tarun Patel is a Dentist practicing in Pacoima, CA. Dr. Patel specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Functional analyses of type V adenylyl cyclase.
- Developmental expression of PAM (protein associated with MYC) in the rodent
- Protein-tyrosine phosphatase-1B (PTP1B) mediates the anti-migratory actions of Sprouty.
- Albumin: a Galpha(s)-specific guanine nucleotide dissociation inhibitor and GTPase activating protein.
- Single transmembrane spanning heterotrimeric g protein-coupled receptors and their signaling cascades.
- Sprouty regulates cell migration by inhibiting the activation of Rac1 GTPase.
- Optimal lysophosphatidic acid-induced DNA synthesis and cell migration but not survival require intact autophosphorylation sites of the epidermal growth factor receptor.
- Histidine residues 912 and 913 in protein associated with Myc are necessary for the inhibition of adenylyl cyclase activity.
- Copper and zinc inhibit Galphas function: a nucleotide-free state of Galphas induced by Cu2+ and Zn2+.
- Regulation of vascular smooth muscle cell proliferation and migration by human sprouty 2.
- The tumor suppressor PTEN is necessary for human Sprouty 2-mediated inhibition of cell proliferation.
- Subcellular localization and biological actions of activated RSK1 are determined by its interactions with subunits of cyclic AMP-dependent protein kinase.
- A historical perspective of the EGF receptor and related systems.
- Analysis of EGF receptor interactions with the alpha subunit of the stimulatory GTP binding protein of adenylyl cyclase, Gs.
- A recombinant transductor-effector system: in vitro study of G inhibitory protein (G-alpha-i1) direct activators.
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