Dr. David Israel Beller DDS
Dentist
809 N Bethlehem Pike Spring House PA, 19477Rating
3/5
About
Dr. David Beller is a Dentist practicing in Spring House, PA. Dr. Beller specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Aberrant macrophage cytokine production is a conserved feature among autoimmune-prone mouse strains: elevated interleukin (IL)-12 and an imbalance in tumor necrosis factor-alpha and IL-10 define a unique cytokine profile in macrophages from young non
- Distinct pathways for NF-kappa B regulation are associated with aberrant macrophage IL-12 production in lupus- and diabetes-prone mouse strains.
- Monocyte chemoattractant protein-1 regulates adhesion molecule expression and cytokine production in human monocytes.
- Thymic maturation in vitro by a secretory product from macrophages.
- Thymic maturation in vitro by a secretory product from macrophages.
- Thymic macrophages modulate one stage of T cell differentiation in vitro.
- Thymic macrophages modulate one stage of T cell differentiation in vitro.
- Imbalanced cytokine production by macrophages from autoimmune-prone mice.
- Regulation of macrophage activation by IL-3. II. IL-3 and lipopolysaccharide act synergistically in the regulation of IL-1 expression.
- Regulation of macrophage activation by IL-3. I. IL-3 functions as a macrophage-activating factor with unique properties, inducing Ia and lymphocyte function-associated antigen-1 but not cytotoxicity.
- Aberrant regulation of IL-1 expression in macrophages from young autoimmune-prone mice.
- Lymphokine-independent induction of macrophage membrane IL-1 by autoreactive T cells recognizing either class I or class II MHC determinants.
- Induction of macrophage membrane interleukin 1 expression by T-cell dependent and T-cell independent pathways is inhibited by cyclosporin A.
- Macrophage activation for antileishmanial defense by an apparently novel mechanism.
- Simultaneous expression of Ia and cytocidal activity by macrophages, and the consequences for antigen presentation.
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