Dr. Christopher Randolph Burrow M.D.
Nephrologist (Kidney Specialist) | Nephrology
1155 Camino Del Mar # 429 Del Mar CA, 92014About
Dr. Christopher Burrow is a nephrologist practicing in Del Mar, CA. Dr. Burrow specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Burrow most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Baylor College of Medicine 1978
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Nephrology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Cystic diseases of the kidney: role of adhesion molecules in normal and abnormal tubulogenesis.
- Identification of phosphorylation sites in the PKD1-encoded protein C-terminal domain.
- The PKD1 gene product, "polycystin-1," is a tyrosine-phosphorylated protein that colocalizes with alpha2beta1-integrin in focal clusters in adherent renal epithelia.
- Apical plasma membrane mispolarization of NaK-ATPase in polycystic kidney disease epithelia is associated with aberrant expression of the beta2 isoform.
- Regulatory molecules in kidney development.
- Monomeric midkine induces tumor cell proliferation in the absence of cell-surface proteoglycan binding.
- Retinoids and renal development.
- Modification of the composition of polycystin-1 multiprotein complexes by calcium and tyrosine phosphorylation.
- PRKX, a phylogenetically and functionally distinct cAMP-dependent protein kinase, activates renal epithelial cell migration and morphogenesis.
- Na transport in autosomal recessive polycystic kidney disease (ARPKD) cyst lining epithelial cells.
- Ethnic differences in cardiac potassium channel variants: implications for genetic susceptibility to sudden cardiac death and genetic testing for congenital long QT syndrome.
- Autosomal dominant polycystic kidney disease: cellular and molecular mechanisms of cyst formation.
- Spectrum and prevalence of cardiac sodium channel variants among black, white, Asian, and Hispanic individuals: implications for arrhythmogenic susceptibility and Brugada/long QT syndrome genetic testing.
- Disruption of polycystin-1 function interferes with branching morphogenesis of the ureteric bud in developing mouse kidneys.
- Protein kinase X activates ureteric bud branching morphogenesis in developing mouse metanephric kidney.
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