Stuart J Shankland
Nephrologist (Kidney Specialist) | Nephrology
University Of Washington Medical Ctr 1959 Ne Pacific St Seattle WA, 98195About
Dr. Shankland is a UW Belding H. Scribner Endowed Chair of Medicine. He is also Professor of Medicine and Head of the Division of Nephrology Clinical Interests Glomerulonephritis Diabetic kidney disease ...
Education and Training
Univ of Cape Town 1983
University of Cape Town Faculty of Health Sciences 1983
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Modulation of apoptosis by the cyclin-dependent kinase inhibitor p27(Kip1).
- Glomerular podocytes and adhesive interaction with glomerular basement membrane.
- Cell cycle regulatory proteins in glomerular disease.
- The cyclin kinase inhibitor p21CIP1/WAF1 limits glomerular epithelial cell proliferation in experimental glomerulonephritis.
- The cyclin kinase inhibitor p21WAF1/CIP1 is required for glomerular hypertrophy in experimental diabetic nephropathy.
- Cyclin kinase inhibitor p21CIP1/WAF1 limits interstitial cell proliferation following ureteric obstruction.
- Cell cycle regulatory proteins in glomerular disease.
- Cell cycle regulatory proteins in renal disease: role in hypertrophy, proliferation, and apoptosis.
- Vascular endothelial growth factor accelerates renal recovery in experimental thrombotic microangiopathy.
- Complement (C5b-9) induces DNA synthesis in rat mesangial cells in vitro.
- High glucose-induced hypertrophy of mesangial cells requires p27(Kip1), an inhibitor of cyclin-dependent kinases.
- Podocyte expression of the CDK-inhibitor p57 during development and disease.
- Cyclooxygenase-2 overexpression inhibits platelet-derived growth factor-induced mesangial cell proliferation through induction of the tumor suppressor gene p53 and the cyclin-dependent kinase inhibitors p21waf-1/cip-1 and p27kip-1.
- Mechanical stress reduces podocyte proliferation in vitro.
Treatments
- Glomerulonephritis, Proteinuria
Fellowships
- University of Toronto, Wellesley Hospital
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