Dr. Bart Clifton Moulton MD
Pulmonologist | Pulmonary Disease
3181 Sw Sam Jackson Park Rd Portland OR, 97239About
Dr. Bart Moulton practices Pulmonology in Portland, OR. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Moulton manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
University of Washington School of Medicine 2005
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Regression of the decidualized mesometrium and decidual cell apoptosis are associated with a shift in expression of Bcl2 family members.
- Progesterone and estrogen control of uterine prostaglandin dehydrogenase activity during deciduomal growth.
- Progestin and estrogen control of cathepsin D expression and processing in rat uterine luminal epithelium and stroma-myometrium.
- Progesterone withdrawal and RU-486 treatment stimulate apoptosis in specific uterine decidual cells.
- Estrogen induced expression of the C-jun proto-oncogene in the immature and mature rat uterus.
- Muscarinic receptor antagonists, from folklore to pharmacology; finding drugs that actually work in asthma and COPD.
- Regulation of 3-hydroxyl-3-methylglutaryl-coenzyme A reductase activity in rat uterine tissues.
- Hormonal control of phospholipid methylation in uterine luminal epithelial cells during uterine sensitivity to deciduogenic stimuli.
- Biochemical responses of the luminal epithelium and uterine sensitization.
- Effects of 8-azaguanine on the induction of uterine glucose-6-phosphate dehydrogenase activity by estradiol or NADP plus.
- Synthesis and degradation of glucose-6-phosphate dehydrogenase in the rat uterus.
- Effects of RNA and protein synthesis inhibitors on preinduced levels of rat uterine glucose-6-phosphate dehydrogenase.
- Progesterone and estrogen control of uterine glucose-6-phosphate dehydrogenase activity during deciduomal growth.
- Ovum implantation and uterine lysosomal enzyme activity.
- A delayed antagonistic effect of progesterone on estradiol induced increases in uterine glucose-6-phosphate dehydrogenase.
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