Dr. Gregory C Mathews MD
Neurologist | Neurology
6410 Rockledge Dr Suite 410 Bethesda MD, 20817About
Dr. Gregory Mathews is a distinguished Neurologist in Bethesda, MD. Dr. Mathews specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Mathews employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Mathews is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Washington University In St Louis School of Medicine,St Louis, Mo, United States
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN- Clinical Neurophysiology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Poststroke seizures.
- Implicating A-current in a hereditary partial epilepsy.
- Evidence for the action of endogenous adenosine in the rabbit retina: modulation of the light-evoked release of acetylcholine.
- The dual roles of GABA in seizures and epilepsy generate more excitement.
- Glutamine uptake by System A transporters maintains neurotransmitter GABA synthesis and inhibitory synaptic transmission.
- A possible explanation for the paradox of hyperexcitability and epilepsy in "loss of function" voltage-gated sodium channel mutations.
- Is too much inhibition to blame in autosomal dominant nocturnal frontal lobe epilepsy?
- Alterations of GABA A-receptor function and allosteric modulation during development of status epilepticus.
- NKCC1 and KCC2 prevent hyperexcitability in the mouse hippocampus.
- Hypermotor seizures in patients with temporal pole lesions.
- In vivo metabotropic glutamate receptor 5 (mGluR5) antagonism prevents cocaine-induced disruption of postsynaptically maintained mGluR5-dependent long-term depression.
- Rational polypharmacy: when two old drugs are better than one.
- Selective changes in hippocampal GABAA receptors during status epilepticus.
- Generalized-onset seizures with secondary focal evolution.
- Glutamate spillover augments GABA synthesis and release from axodendritic synapses in rat hippocampus.
Dr. Gregory C Mathews MD's Practice location
SILVER SPRING, MD 20910Get Direction
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