Dr. Kenneth Kai Cheng DDS
Oral and Maxillofacial Surgeon | Oral and Maxillofacial Surgery
385 Tremont Ave East Orange NJ, 07018About
Dr. Kenneth Cheng is an oral and maxillofacial surgeon practicing in East Orange, NJ. Dr. Cheng specializes in the treatment of problems related to the face, mouth and jaws. As an oral and maxillofacial surgeon, Dr. Cheng is a unique dental specialist who can provide emergency medicine, perform general surgery and give anesthesia. These medical doctors are the only type of medical care specialist who can administer anesthesia, besides anesthesiologists. Typical procedures performed by Dr. Cheng are tooth extractions, especially wisdom teeth, corrective jaw surgery, cleft palate surgery and reconstructive surgery after an injury. Oral and maxillofacial surgeons can also perform dental work including placing dental implants. These surgeons might also deal with conditions of sleep apnea, oral cancers and more.
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Data provided by the National Library of Medicine- Adiponectin-induced endothelial nitric oxide synthase activation and nitric oxide production are mediated by APPL1 in endothelial cells.
- Perforation of the soft palate using the GlideScope videolaryngoscope.
- Suppression of the Raf/MEK/ERK signaling cascade and inhibition of angiogenesis by the carboxyl terminus of angiopoietin-like protein 4.
- APPL1 potentiates insulin-mediated inhibition of hepatic glucose production and alleviates diabetes via Akt activation in mice.
- Adiponectin prevents diabetic premature senescence of endothelial progenitor cells and promotes endothelial repair by suppressing the p38 MAP kinase/p16INK4A signaling pathway.
- APPL1 counteracts obesity-induced vascular insulin resistance and endothelial dysfunction by modulating the endothelial production of nitric oxide and endothelin-1 in mice.
- APPL1 potentiates insulin secretion in pancreatic β cells by enhancing protein kinase Akt-dependent expression of SNARE proteins in mice.
- PICK1 and ICA69 control insulin granule trafficking and their deficiencies lead to impaired glucose tolerance.
- TRAF6-mediated ubiquitination of APPL1 enhances hepatic actions of insulin by promoting the membrane translocation of Akt.
- APPL1 transgenic mice are protected from high-fat diet-induced cardiac dysfunction.
- Signaling mechanisms underlying the insulin-sensitizing effects of adiponectin.
- The adaptor protein APPL2 inhibits insulin-stimulated glucose uptake by interacting with TBC1D1 in skeletal muscle.
- NUCKS is a positive transcriptional regulator of insulin signaling.
- The MDM2-p53-pyruvate carboxylase signalling axis couples mitochondrial metabolism to glucose-stimulated insulin secretion in pancreatic β-cells.
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