Dr. Philip W Askenase MD
Allergist and Immunologist
789 Howard Ave Dana Bldg - 3rd Fl New Haven CT, 06519About
Dr. Philip Askenase practices Allergy and Immunology care in New Haven, CT. Dr. Askenase specializes in the diagnosis and treatment of asthma and other allergic diseases. Allergist-Immunologists are trained and certified to treat each patients sensitivity and response to allergens of varying severity. Dr. Askenase provides several means of testing and treatment to increase immunity to potentially harmful substances.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Human allogeneic vascular rejection after arterial transplantation and peripheral lymphoid reconstitution in severe combined immunodeficient mice.
- A new paradigm of T cell allergy: requirement for the B-1 cell subset.
- Role of interleukin-4 in down-regulation of contact sensitivity by gammadelta T cells from tolerized T-cell receptor alpha-/- mice.
- Endothelial cell E- and P-selectin up-regulation in murine contact sensitivity is prolonged by distinct mechanisms occurring in sequence.
- IL-12 is produced by antigen-presenting cells stimulated with soluble alphabeta TCR and restores impaired T(h)1 responses.
- Cutaneous basophil hypersensitivity uncovered in the cell transfer of classical tuberculin hypersensitivity.
- Gee whiz: CpG DNA allergy therapy!
- Early local generation of C5a initiates the elicitation of contact sensitivity by leading to early T cell recruitment.
- IL-12 reverses established tolerance mediated by TCRalphabeta+ but not by TCRgammadelta+ suppressor T cells.
- Atypical fatal hypocomplementemic urticarial vasculitis with involvement of native and homograft aortic valves in an African American man.
- Basophil responses to chemokines are regulated by both sequential and cooperative receptor signaling.
- Allergen-specific Th1 cells counteract efferent Th2 cell-dependent bronchial hyperresponsiveness and eosinophilic inflammation partly via IFN-gamma.
- B-1 B cell IgM antibody initiates T cell elicitation of contact sensitivity.
- T cytotoxic 1 and T cytotoxic 2 CD8 T cells both inhibit IgE responses.
- Specific basophil hypersensitivity induced by skin testing and transferred using immune serum.
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