Dr. Rosa Maria Soler DDS
Dentist | General Practice
4240 W 16th Ave Hialeah FL, 33012Rating
5/5
About
Dr. Rosa Soler is a Dentist practicing in Hialeah, FL. Dr. Soler specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Activation of phosphatidylinositol 3-kinase, but not extracellular-regulated kinases, is necessary to mediate brain-derived neurotrophic factor-induced motoneuron survival.
- Receptors of the glial cell line-derived neurotrophic factor family of neurotrophic factors signal cell survival through the phosphatidylinositol 3-kinase pathway in spinal cord motoneurons.
- Nerve growth factor activation of the extracellular signal-regulated kinase pathway is modulated by Ca(2+) and calmodulin.
- Neuronal survival induced by neurotrophins requires calmodulin.
- Cytokines promote motoneuron survival through the Janus kinase-dependent activation of the phosphatidylinositol 3-kinase pathway.
- Glial cell line-derived neurotrophic factor increases intracellular calcium concentration. Role of calcium/calmodulin in the activation of the phosphatidylinositol 3-kinase pathway.
- Pyruvate protects cerebellar granular cells from 6-hydroxydopamine-induced cytotoxicity by activating the Akt signaling pathway and increasing glutathione peroxidase expression.
- Neuroprotection by neurotrophic factors and membrane depolarization is regulated by calmodulin kinase IV.
- Tyr-701 is a new regulatory site for neurotrophin receptor TrkA trafficking and function.
- Tumor necrosis factor alpha and interferon gamma cooperatively induce oxidative stress and motoneuron death in rat spinal cord embryonic explants.
- Specific vulnerability of mouse spinal cord motoneurons to membrane depolarization.
- The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins.
- A new model to study spinal muscular atrophy: neurite degeneration and cell death is counteracted by BCL-X(L) Overexpression in motoneurons.
- The canonical nuclear factor-κB pathway regulates cell survival in a developmental model of spinal cord motoneurons.
- SMN deficiency attenuates migration of U87MG astroglioma cells through the activation of RhoA.
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Nearest Hospitals
LARKIN COMMUNITY HOSPITAL PALM SPRINGS CAMPUSl
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2001 W 68TH ST HIALEAH FL 33016