Dr. William A Pulsinelli PHD, MD
Neurologist | Neurology
1211 Union Ave Suite 200 Memphis TN, 38104About
Dr. William Pulsinelli is a distinguished Neurologist in Memphis, TN. Dr. Pulsinelli specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Pulsinelli employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Pulsinelli is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
University of Utah School of Medicine 1973
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Changes in membrane properties of CA1 pyramidal neurons after transient forebrain ischemia in vivo.
- Transient global ischemia in rats yields striatal projection neuron and interneuron loss resembling that in Huntington's disease.
- Switch in glutamate receptor subunit gene expression in CA1 subfield of hippocampus following global ischemia in rats.
- Hypothermic protection in rat focal ischemia models: strain differences and relevance to "reperfusion injury".
- Depolarization thresholds for hippocampal damage, ischemic preconditioning, and changes in gene expression after global ischemia in the rat.
- Failure of GM1 ganglioside to influence outcome in experimental focal ischemia.
- The CBF threshold and dynamics for focal cerebral infarction in spontaneously hypertensive rats.
- Cerebral blood flow thresholds for mRNA synthesis after focal ischemia and the effect of MK-801.
- Blockade of the AMPA receptor prevents CA1 hippocampal injury following severe but transient forebrain ischemia in adult rats.
- Transient cooling during early reperfusion attenuates delayed edema and infarct progression in the Spontaneously Hypertensive Rat. Distribution and time course of regional brain temperature change in a model of postischemic hypothermic protection.
- Acid-induced death in neurons and glia.
- Dynamics of interstitial and intracellular pH in evolving brain infarct.
- The N-methyl-D-aspartate antagonist, MK-801, fails to protect against neuronal damage caused by transient, severe forebrain ischemia in adult rats.
- Metabolic and perfusion responses to recurrent peri-infarct depolarization during focal ischemia in the Spontaneously Hypertensive Rat: dominant contribution of sporadic CBF decrements to infarct expansion.
- Nimodipine pretreatment improves cerebral blood flow and reduces brain edema in conscious rats subjected to focal cerebral ischemia.
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Practice At 1211 Union Ave Suite 200
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METHODIST HEALTHCARE MEMPHIS HOSPITALSl
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877 JEFFERSON AVENUE MEMPHIS TN 38103