Dr. Ronald Lynn Wilder M.D.
Rheumatologist | Rheumatology
10319 Westlake Dr Suite 213 Bethesda MD, 20817About
Dr. Ronald Wilder is a rheumatologist practicing in Bethesda, MD. Dr. Wilder specializes in the treatment of musculoskeletal diseases and systematic autoimmune conditions that can affect the bones, muscles or bones. Eventually, if not treated, these illnesses can also impact the skin, eyes, nervous system and internal organs. Dr. Wilder treats diseases similar to orthopedists but does not perform surgery. Often times, research is conducted to find potential alternatives for the patients illness.
Education and Training
University of California 1974
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Rheumatology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Evaluation of quantitative trait loci regulating severity of mycobacterial adjuvant-induced arthritis in monocongenic and polycongenic rats: identification of a new regulatory locus on rat chromosome 10 and evidence of overlap with rheumatoid arthrit
- Mutation of macrophage colony stimulating factor (Csf1) causes osteopetrosis in the tl rat.
- Neuroimmunoendocrinology of the rheumatic diseases: past, present, and future.
- Application of interval haplotype analysis facilitates efficient mapping of the mutation causing osteopetrosis in tl rats.
- Modulation of multiple experimental arthritis models by collagen-induced arthritis quantitative trait loci isolated in congenic rat lines: different effects of non-major histocompatibility complex quantitative trait loci in males and females.
- Localization of the mutation responsible for osteopetrosis in the op rat to a 1.5-cM genetic interval on rat chromosome 10: identification of positional candidate genes by radiation hybrid mapping.
- Pilot clinical trial of intravenous doxycycline versus placebo for rheumatoid arthritis.
- Identification of two novel female-specific non-major histocompatibility complex loci regulating collagen-induced arthritis severity and chronicity, and evidence of epistasis.
- The non-major histocompatibility complex quantitative trait locus Cia10 contains a major arthritis gene and regulates disease severity, pannus formation, and joint damage.
- The non-MHC quantitative trait locus Cia5 contains three major arthritis genes that differentially regulate disease severity, pannus formation, and joint damage in collagen- and pristane-induced arthritis.
- Low- versus high-baseline epinephrine output shapes opposite innate cytokine profiles: presence of Lewis- and Fischer-like neurohormonal immune phenotypes in humans?
- Identification of two new arthritis severity loci that regulate levels of autoantibodies, interleukin-1β, and joint damage in pristane- and collagen-induced arthritis.
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