Dr. Edward J Mcmanus M.D.
Infectious Disease Specialist | Infectious Disease
765 Route 10 East Randolph NJ, 07869About
Dr. Edward Mcmanus is an infectious disease specialist practicing in Randolph, NJ. Dr. Mcmanus specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Infectious Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- TSC1-TSC2: a complex tale of PKB-mediated S6K regulation.
- The in vivo role of PtdIns(3,4,5)P3 binding to PDK1 PH domain defined by knockin mutation.
- Cancer, oncogenes and signal transduction.
- Role that phosphorylation of GSK3 plays in insulin and Wnt signalling defined by knockin analysis.
- Role of the PDK1-PKB-GSK3 pathway in regulating glycogen synthase and glucose uptake in the heart.
- A novel regulation of IRS1 (insulin receptor substrate-1) expression following short term insulin administration.
- Analysis of hepatic gene transcription in mice expressing insulin-insensitive GSK3.
- Distinct priming kinases contribute to differential regulation of collapsin response mediator proteins by glycogen synthase kinase-3 in vivo.
- The structure of SOCS3 reveals the basis of the extended SH2 domain function and identifies an unstructured insertion that regulates stability.
- SOCS-3 negatively regulates innate and adaptive immune mechanisms in acute IL-1-dependent inflammatory arthritis.
- Cdc42 and noncanonical Wnt signal transduction pathways cooperate to promote cell polarity.
- Mechanism of crosstalk inhibition of IL-6 signaling in response to LPS and
- Polycomb repressive complex 2 (PRC2) restricts hematopoietic stem cell activity.
- Deletion of the SOCS box of suppressor of cytokine signaling 3 (SOCS3) in embryonic stem cells reveals SOCS box-dependent regulation of JAK but not STAT phosphorylation.
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