Dr. Michael David Weiden M.D.
Critical Care Surgeon | Critical Care Medicine
550 First Ave Nbh8e32 New York NY, 10016About
Dr. Michael Weiden is a critical care surgeon practicing in New York, NY. Dr. Weiden specializes in the needs of critically ill surgical patients. Critical care surgeons are experts in treating physiologic responses to tissue injury from trauma, burns, infections, acute inflammation, operation and more. They are also trained to know how such injuries interact with other diseases that a patient may have. As a critical care surgeon, Dr. Weiden has a broad knowledge base and full understanding of the biology of the critically ill patient and the patients organ system functions.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Aberrant expression of immunoglobulin heavy chain genes in Epstein-Barr virus-negative, human immunodeficiency virus-related lymphoid interstitial pneumonia.
- Oligoclonal T cell expansions in pulmonary lymphoproliferative disorders: demonstration of the frequent occurrence of oligoclonal T cells in human immunodeficiency virus-related lymphoid interstitial pneumonia.
- CD40 contributes to lethality in acute sepsis: in vivo role for CD40 in innate immunity.
- Surfactant protein A modulates the inflammatory response in macrophages during tuberculosis.
- Aerosolized gamma interferon (IFN-gamma) induces expression of the genes encoding the IFN-gamma-inducible 10-kilodalton protein but not inducible nitric oxide synthase in the lung during tuberculosis.
- Exogenous gamma and alpha/beta interferon rescues human macrophages from cell death induced by Bacillus anthracis.
- Rhodococcus equi endobronchial mass with lung abscess in a patient with AIDS.
- Cd40 but not CD154 knockout mice have reduced inflammatory response in polymicrobial sepsis: a potential role for Escherichia coli heat shock protein 70 in CD40-mediated inflammation in vivo.
- Vascular endothelial growth factor blockade reduces plasma cytokines in a murine model of polymicrobial sepsis.
- Exogenous interferon-alpha and interferon-gamma increase lethality of murine inhalational anthrax.
- Gene expression profiles of bronchoalveolar cells in pulmonary TB.
- Integration of HIV-1 caused STAT3-associated B cell lymphoma in an AIDS patient.
- Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis.
- Emerging exposures and respiratory health: World Trade Center dust.
- Neutrophils activate alveolar macrophages by producing caspase-6-mediated cleavage of IL-1 receptor-associated kinase-M.
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