Dr. Jack A Elias MD
Pulmonologist | Pulmonary Disease
789 Howard Ave Fitkin Building - 2n New Haven CT, 06519About
Dr. Jack Elias practices Pulmonology in New Haven, CT. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Elias manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
Perelman School of Medicine University of Pennsylvania 1976
Provider Details
Expert Publications
Data provided by the National Library of Medicine- IL-13-induced chemokine responses in the lung: role of CCR2 in the pathogenesis of IL-13-induced inflammation and remodeling.
- Pulmonary type II cell hypertrophy and pulmonary lipoproteinosis are features of chronic IL-13 exposure.
- Transgenic overexpression of interleukin (IL)-10 in the lung causes mucus metaplasia, tissue inflammation, and airway remodeling via IL-13-dependent and -independent pathways.
- Tetracycline-controlled transcriptional regulation systems: advances and application in transgenic animal modeling.
- Overlapping and enzyme-specific contributions of matrix metalloproteinases-9 and -12 in IL-13-induced inflammation and remodeling.
- Interleukin-13 induces surfactant function abnormality in the murine lung.
- Interleukin-13 and leukotrienes: an intersection of pathogenetic schema.
- Cytokine regulation of IL-13Ralpha2 and IL-13Ralpha1 in vivo and in vitro.
- Polarized in vivo expression of IL-11 and IL-17 between acute and chronic skin lesions.
- Interleukin-11 and interleukin-6 protect cultured human endothelial cells from H2O2-induced cell death.
- Pulmonary expression of leukemia inhibitory factor induces B cell hyperplasia and confers protection in hyperoxia.
- Adenosine mediates IL-13-induced inflammation and remodeling in the lung and interacts in an IL-13-adenosine amplification pathway.
- The C10/CCL6 chemokine and CCR1 play critical roles in the pathogenesis of IL-13-induced inflammation and remodeling.
- Early growth response gene 1-mediated apoptosis is essential for transforming growth factor beta1-induced pulmonary fibrosis.
Dr. Jack A Elias MD's Practice location
New Haven, CT 06519Get Direction
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