Dr. Peter Andrija Nigrovic MD
Rheumatologist (Pediatric) | Pediatric Rheumatology
ONE JIMMY FUND WAY BOSTON MA, 02115About
Dr. Peter Nigrovic practices Pediatric Rheumatology in BOSTON, MA. Pediatric rheumatologists treat child patients who suffer from pain in the musculoskeletal system, have symptoms of arthritis, or an autoimmune disorder. Dr. Nigrovic evaluates and treats a variety of joint, muscle, and bone disorders; including autoimmune disorders, such as lupus, juvenile rheumatoid arthritis, scleroderma, Kawasaki disease, post-infectious arthritis, chronic vasculitis, and inflammatory disorders of the muscle, eye, or other organs. Dr. Nigrovic also is trained in the evaluation of prolonged fever, unexplained complaints of chronic musculoskeletal pain, weakness, poor appetite, fatigue, and/or loss of function or skills.
Education and Training
Harvard Med Sch, Boston Ma MD
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mast cells in inflammatory arthritis.
- Particularities of the vasculature can promote the organ specificity of autoimmune attack.
- Mast cells in autoantibody responses and arthritis.
- Patients with juvenile psoriatic arthritis comprise two distinct populations.
- Subpopulations within juvenile psoriatic arthritis: a review of the literature.
- Synovial mast cells: role in acute and chronic arthritis.
- Comparison of Vancouver and International League of Associations for rheumatology classification criteria for juvenile psoriatic arthritis.
- Juvenile psoriatic arthritis: bathwater or baby?
- Neutrophils in a mouse model of autoantibody-mediated arthritis: critical producers of Fc receptor gamma, the receptor for C5a, and lymphocyte function-associated antigen 1.
- C5a receptor enables participation of mast cells in immune complex arthritis independently of Fcγ receptor modulation.
- Hypogalactosylation of serum N-glycans fails to predict clinical response to methotrexate and TNF inhibition in rheumatoid arthritis.
- Platelets can enhance vascular permeability.
- Multiple juvenile idiopathic arthritis subtypes demonstrate proinflammatory IgG glycosylation.
- Ly6G ligation blocks recruitment of neutrophils via a β2-integrin-dependent mechanism.
- Platelets: active players in the pathogenesis of arthritis and SLE.
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