Dr. Hussein D Foda MD
Pulmonologist | Pulmonary Disease
Northport Vamc 79 Middleville Rd Northport NY, 11768About
Dr. Hussein Foda practices Pulmonology in Northport, NY. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Foda manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
University Of Cairo Faculty Of Medicine 1979
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Attenuation of oxidant-induced lung injury by the synthetic matrix metalloproteinase inhibitor BB-3103.
- Tumorigenic potential of extracellular matrix metalloproteinase inducer.
- Ventilator-induced lung injury upregulates and activates gelatinases and EMMPRIN: attenuation by the synthetic matrix metalloproteinase inhibitor, Prinomastat (AG3340).
- Mechanical stretch induces MMP-2 release and activation in lung endothelium: role of EMMPRIN.
- Nitric oxide and vasoactive intestinal peptide as co-transmitters of airway smooth-muscle relaxation: analysis in neuronal nitric oxide synthase knockout mice.
- [Pulmonary apoptosis and necrosis in hyperoxia-induced acute mouse lung injury].
- CXCR4 and CXCL12 (SDF-1) in prostate cancer: inhibitory effects of human single chain Fv antibodies.
- Cyclic mechanical strain-induced proliferation and migration of human airway smooth muscle cells: role of EMMPRIN and MMPs.
- Oxytocin and related peptides elicit contractions of prostate and seminal vesicle.
- [Expression of various matrix metalloproteinases in mice with hyperoxia-induced acute lung injury].
- [Met]enkephalin-Arg6-Gly7-Leu8-immunoreactive nerves in guinea-pig and rat lungs: distribution, origin, and co-existence with vasoactive intestinal polypeptide immunoreactivity.
- [Study on the function of osteopontin in hyperoxia-induced acute lung injury and its mechanism].
- Angiogenesis is induced by airway smooth muscle strain.
- [The role of disequilibrium of expression of matrix metalloproteinase-2/9 and their tissue inhibitors in pathogenesis of hyperoxia-induced acute lung injury in mice].
- Osteopontin protects against hyperoxia-induced lung injury by inhibiting nitric oxide synthases.
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