Dr. Theodore G Gabig M.D.
Hematologist-Oncologist | Hematology & Oncology
University Hospital. L5 Stony Brook NY, 11794About
Dr. Theodore Gabig is a hematologist oncologist practicing in Stony Brook, NY. Dr. Gabig specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
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Expert Publications
Data provided by the National Library of Medicine- Combined heterozygosity of factor V leiden and the G20210A prothrombin gene mutation in a patient with cerebral cortical vein thrombosis.
- Prevention of arterial thromboembolism in nonvalvular atrial fibrillation: validating molecular markers of risk stratification.
- Expression of Clostridium perfringens enterotoxin receptors claudin-3 and claudin-4 in prostate cancer epithelium.
- Clostridium perfringens enterotoxin elicits rapid and specific cytolysis of breast carcinoma cells mediated through tight junction proteins claudin 3 and 4.
- Deactivation of the subcellular NADPH oxidase and its relationship to termination of the respiratory burst.
- Expression of val-12 mutant ras p21 in an IL-3-dependent murine myeloid cell line is associated with loss of serum-dependence and increases in membrane PIP2-specific phospholipase C activity.
- MOZ and MOZ-CBP cooperate with NF-kappaB to activate transcription from NF-kappaB-dependent promoters.
- Resolution of a low molecular weight G protein in neutrophil cytosol required for NADPH oxidase activation and reconstitution by recombinant Krev-1 protein.
- Growth factor-dependent initiation of DNA replication in nuclei isolated from an interleukin 3-dependent murine myeloid cell line.
- A neutrophil GTP-binding protein that regulates cell free NADPH oxidase activation is located in the cytosolic fraction.
- Purification and characterization of a lipid thiobis ester from human neutrophil cytosol that reversibly deactivates the O2- -generating NADPH oxidase.
- A RAS oncogene imparts growth factor independence to myeloid cells that abnormally regulate protein kinase C: a nonautocrine transformation pathway.
- Reversible activation of the neutrophil superoxide generating system by hexachlorocyclohexane: correlation with effects on a subcellular superoxide-generating fraction.
- A claudin 3 and claudin 4-targeted Clostridium perfringens protoxin is selectively cytotoxic to PSA-producing prostate cancer cells.
- A mutant RAS gene acts through protein kinase C to augment interleukin-3 dependent proliferation in a fastidious immortal myeloid cell line.
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Nearby Providers
- Dr. Sandy Anne M.D.Stony Brook University Hospital Stony Brook NY 11794
- Dr. Jules A Cohen M.D.Stony Brook Cancer Ctr Stony Brook NY 11794
- Dr. Huda Salman M.D.HSC T15 040 STONY BROOK NY 11794
- Ms. Anu Radha Neerukonda M.D.Hsc T16 020 Stony Brook NY 11794
- Dr. Min Janice Lu M.D.University Hospital, L5 Stony Brook NY 11794
- Dr. Roger S. Keresztes MD3 Edmund Pellegrino Rd Stony Brook NY 11794