Dr. Grant Gordon Kelley M.D.
Hospitalist
301 Prospect Ave Hospital Internists Syracuse NY, 13203About
Dr. Grant Kelley is a hospitalist practicing in Syracuse, NY. Dr. Kelley specializes in the comprehensive medical care of hospitalized patients. As a hospitalist, Dr. Kelley manages the clinical problems of hospitalized patients and the acutely ill, while working to improve the performance of the hospital. Dr. Kelley works in collaboration with all of the different doctors that are working with the patient. Hospitalists are involved in the diagnosis, treatment and medical procedures of patients.
Education and Training
University of Saskatchewan / College of Medicine 1987
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Phospholipase C(epsilon): a novel Ras effector.
- Fuel and hormone regulation of phospholipase C beta 1 and delta 1 overexpressed in RINm5F pancreatic beta cells.
- Activation of phospholipase C-epsilon by heterotrimeric G protein betagamma-subunits.
- Involvement of phosphatidylinositol 3-kinase, but not RalGDS, in TC21/R-Ras2-mediated transformation.
- Further studies on the growth of human tissue cells in the absence of an added gas phase.
- The isolation and propagation of human epidermoid carcinoma cells resistant to 6-mercaptopurine.
- The infection of chickembryo fibroblasts in suspension culture by the Rous sarcoma virus.
- Purification of phospholipase C beta and phospholipase C epsilon from Sf9 cells.
- Hormonal regulation of phospholipase Cepsilon through distinct and overlapping pathways involving G12 and Ras family G-proteins.
- Involvement of the p97-Ufd1-Npl4 complex in the regulated endoplasmic reticulum-associated degradation of inositol 1,4,5-trisphosphate receptors.
- Phospholipase C epsilon modulates beta-adrenergic receptor-dependent cardiac contraction and inhibits cardiac hypertrophy.
- Genetic and pharmacologic dissection of Ras effector utilization in oncogenesis.
- Epac-mediated activation of phospholipase C(epsilon) plays a critical role in beta-adrenergic receptor-dependent enhancement of Ca2+ mobilization in cardiac myocytes.
- SPFH2 mediates the endoplasmic reticulum-associated degradation of inositol 1,4,5-trisphosphate receptors and other substrates in mammalian cells.
- Phospholipase Cepsilon is a nexus for Rho and Rap-mediated G protein-coupled receptor-induced astrocyte proliferation.
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