Dr. Michael George Rosenberg
Infectious Disease Specialist (Pediatric) | Pediatric Infectious Diseases
1400 PELHAM PKWY S BRONX NY, 10461About
Dr. Michael Rosenberg is a pediatric infectious disease specialist practicing in BRONX, NY. Dr. Rosenberg specializes in recurring or persistent diseases caused by bacteria, parasites or fungus in infants, children and adolescents. Pediatric infectious disease specialists also provide consultation to other health care professionals dealing with complex cases.
Education and Training
Univ Of Belgrade- Med Fak- Beograd- Serbia 1980
Baylor College of Medicine, Houston, Texas 1987
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Effect of supramolecular inclusion on the selectivity of 3-nortricyclanylidene.
- Selective loss of innate CD4(+) V alpha 24 natural killer T cells in human immunodeficiency virus infection.
- HIV-specific CD8+ T cell function in children with vertically acquired HIV-1 infection is critically influenced by age and the state of the CD4+ T cell compartment.
- Expression of IgA and IgM Fc receptors on murine T lymphocytes.
- Expansion of CD7(low) and CD7(negative) CD8 T-cell effector subsets in HIV-1 infection: correlation with antigenic load and reversion by antiretroviral treatment.
- Partial treatment interruption of protease inhibitors augments HIV-specific immune responses in vertically infected pediatric patients.
- Higher frequency of HIV-1-specific T cell immune responses in African American children vertically infected with HIV-1.
- CD8 T cell effector maturation in HIV-1-infected children.
- Partial treatment interruption of protease inhibitor-based highly active antiretroviral therapy regimens in HIV-infected children.
- Strong HIV-1-specific T cell responses in HIV-1-exposed uninfected infants and neonates revealed after regulatory T cell removal.
- Natural killer cells in perinatally HIV-1-infected children exhibit less degranulation compared to HIV-1-exposed uninfected children and their expression of KIR2DL3, NKG2C, and NKp46 correlates with disease severity.
- Single-cell, phosphoepitope-specific analysis demonstrates cell type- and pathway-specific dysregulation of Jak/STAT and MAPK signaling associated with in vivo human immunodeficiency virus type 1 infection.
- Suppression of HIV-1 plasma viral load below detection preserves IL-17 producing T cells in HIV-1 infection.
- Lower cytokine secretion ex vivo by natural killer T cells in HIV-infected individuals is associated with higher CD161 expression.
- Cross-sectional dating of novel haplotypes of HERV-K 113 and HERV-K 115 indicate these proviruses originated in Africa before Homo sapiens.
Treatments
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