Dr. Ronald J Koenig MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
1500 East Medical Center Dr 1st Floor Cancer & G Ann Arbor MI, 48109About
Dr. Ronald Koenig practices Endocrinology in Ann Arbor, MI. Dr. Koenig specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Koenig examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Retinoic acid repression of bone morphogenetic protein 4 in inner ear development.
- Ubiquitinated deiodinase: not dead yet.
- An RNA-binding domain in the thyroid hormone receptor enhances transcriptional
- A method for efficient production of recombinant thyroid hormone receptors reveals that receptor homodimer-DNA binding is enhanced by the coactivator TIF2.
- Regulation of type 1 iodothyronine deiodinase in health and disease.
- Regulation of thyroid hormone receptor alpha2 RNA binding and subcellular localization by phosphorylation.
- Induction of type 1 iodothyronine deiodinase to prevent the nonthyroidal illness syndrome in mice.
- Delineation, functional validation, and bioinformatic evaluation of gene expression in thyroid follicular carcinomas with the PAX8-PPARG translocation.
- Variable RXR requirements for thyroid hormone responsiveness of endogenous genes.
- The role of bone morphogenetic protein 4 in inner ear development and function.
- Podocyte specific knock out of selenoproteins does not enhance nephropathy in streptozotocin diabetic C57BL/6 mice.
- Modeling the nonthyroidal illness syndrome.
- A locally secreted thyrotropin variant may regulate thyroid function in thyroid inflammatory disorders.
- Dax-1 and steroid receptor RNA activator (SRA) function as transcriptional coactivators for steroidogenic factor 1 in steroidogenesis.
- Paired box gene 8-peroxisome proliferator-activated receptor-gamma fusion protein and loss of phosphatase and tensin homolog synergistically cause thyroid hyperplasia in transgenic mice.
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