Dr. Thomas F Gajewski MD
Hematologist-Oncologist | Hematology & Oncology
180 Harvester Dr Suite 110 Willowbrook IL, 60527About
Dr. Thomas Gajewski is a hematologist oncologist practicing in Willowbrook, IL. Dr. Gajewski specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Interleukin-12-secreting human papillomavirus type 16-transformed cells provide a potent cancer vaccine that generates E7-directed immunity.
- The immune system as anti-tumor sentinel: molecular requirements for an anti-tumor immune response.
- Polymyositis with respiratory muscle weakness requiring mechanical ventilation in a patient with metastatic thymoma treated with octreotide.
- Cutting edge: differentiation of antitumor CTL in vivo requires host expression of Stat1.
- Monitoring specific T-cell responses to melanoma vaccines: ELISPOT, tetramers, and beyond.
- Biochemical analysis of activated T lymphocytes. Protein phosphorylation and Ras,
- Blockade of T cell activation using a surface-linked single-chain antibody to CTLA-4 (CD152).
- Cutting edge: spontaneous rejection of poorly immunogenic P1.HTR tumors by Stat6-deficient mice.
- Transgenic expression of the coxsackie/adenovirus receptor enables adenoviral-mediated gene delivery in naive T cells.
- Absence of CTLA-4 lowers the activation threshold of primed CD8+ TCR-transgenic T cells: lack of correlation with Src homology domain 2-containing protein tyrosine phosphatase.
- Immunization of HLA-A2+ melanoma patients with MAGE-3 or MelanA peptide-pulsed autologous peripheral blood mononuclear cells plus recombinant human interleukin 12.
- Epitope spreading upon P815 tumor rejection triggered by vaccination with the single class I MHC-restricted peptide P1A.
- Antigen-specific blockade of T cells in vivo using dimeric MHC peptide.
- CD28 is not required for c-Jun N-terminal kinase activation in T cells.
- Cutting edge: targeted ligation of CTLA-4 in vivo by membrane-bound anti-CTLA-4 antibody prevents rejection of allogeneic cells.
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