Dr. James Franklin Hatcher M.D., F.A.C.E.P.
Emergency Physician
821 N Cobb St Milledgeville GA, 31061About
Dr. James Hatcher practices Emergency Medicine in Milledgeville, GA. Dr. Hatcher assesses patients who seek immediate medical attention at any time of day or night. Emergency Medicine Physicians are trained to efficiently work with each patient and situation no matter how acute or life-threatening. Dr. Hatcher examines patients, determines means of testing, diagnoses conditions, and decides the best treatment methods.
Education and Training
Howard University College of Medicine 1968
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Arachidonic acid and leukotriene C4: role in transient cerebral ischemia of gerbils.
- CDP-choline: neuroprotection in transient forebrain ischemia of gerbils.
- Elevated N1-acetylspermidine levels in gerbil and rat brains after CNS injury.
- Neuroprotection by group I metabotropic glutamate receptor antagonists in forebrain ischemia of gerbil.
- Lipid alterations in transient forebrain ischemia: possible new mechanisms of CDP-choline neuroprotection.
- Does CDP-choline modulate phospholipase activities after transient forebrain ischemia?
- Effects of citicoline on phospholipid and glutathione levels in transient cerebral ischemia.
- Citicoline: neuroprotective mechanisms in cerebral ischemia.
- Identification of new DNA adducts in human bladder epithelia exposed to the proximate metabolite of 4-aminobiphenyl using 32P-postlabeling method.
- Polyamines and central nervous system injury: spermine and spermidine decrease
- Identification of N-(deoxyguanosin-8-yl)-4-azobiphenyl by (32)P-postlabeling analyses of DNA in human uroepithelial cells exposed to proximate metabolites of the environmental carcinogen 4-aminobiphenyl.
- Citicoline mechanisms and clinical efficacy in cerebral ischemia.
- Soil health index in remediation of contaminated sites. Approach and application.
- Ecotoxicological endpoints for contaminated site remediation.
- Loss of cardiolipin and mitochondria during programmed neuronal death: evidence of a role for lipid peroxidation and autophagy.
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