Dr. Peter Cawood Butler MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
200 Medical Plaza #365 Los Angeles CA, 90095Rating
3/5
About
Dr. Peter Butler practices Endocrinology in Los Angeles, CA. Dr. Butler specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Butler examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Pulsatile insulin secretion by human pancreatic islets.
- Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes.
- Glucose stimulates pulsatile insulin secretion from human pancreatic islets by increasing secretory burst mass: dose-response relationships.
- Replication increases beta-cell vulnerability to human islet amyloid polypeptide-induced apoptosis.
- Diazoxide attenuates glucose-induced defects in first-phase insulin release and pulsatile insulin secretion in human islets.
- Increased beta-cell apoptosis prevents adaptive increase in beta-cell mass in mouse model of type 2 diabetes: evidence for role of islet amyloid formation rather than direct action of amyloid.
- Increased risk of type 2 diabetes in Alzheimer disease.
- Induction of beta-cell rest by a Kir6.2/SUR1-selective K(ATP)-channel opener preserves beta-cell insulin stores and insulin secretion in human islets cultured at high (11 mM) glucose.
- Diabetes due to a progressive defect in beta-cell mass in rats transgenic for human islet amyloid polypeptide (HIP Rat): a new model for type 2 diabetes.
- Pulsatile insulin secretion dictates systemic insulin delivery by regulating
- Activation of peroxisome proliferator-activated receptor-gamma by rosiglitazone protects human islet cells against human islet amyloid polypeptide toxicity by a phosphatidylinositol 3'-kinase-dependent pathway.
- Relationship between beta-cell mass and fasting blood glucose concentration in humans.
- The potential for stem cell therapy in diabetes.
- Postprandial suppression of glucagon secretion depends on intact pulsatile insulin secretion: further evidence for the intraislet insulin hypothesis.
- Hyperinsulinemic hypoglycemia after gastric bypass surgery is not accompanied by islet hyperplasia or increased beta-cell turnover.
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Practice At 200 Medical Plaza #365
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