Dr. Timothy A Sutton M.D.
Nephrologist (Kidney Specialist) | Nephrology
950 W Walnut St E 276 Indianapolis IN, 46202About
Dr. Timothy Sutton is a nephrologist practicing in Indianapolis, IN. Dr. Sutton specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Sutton most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
In Univ Sch of Med, Indianapolis In 1992
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Endothelial injury and dysfunction in ischemic acute renal failure.
- Endothelial injury and dysfunction: role in the extension phase of acute renal failure.
- Minocycline reduces renal microvascular leakage in a rat model of ischemic renal injury.
- Imaging vascular pathology.
- Acute and chronic microvascular alterations in a mouse model of ischemic acute kidney injury.
- Preservation of peritubular capillary endothelial integrity and increasing pericytes may be critical to recovery from postischemic acute kidney injury.
- Live-animal imaging of renal function by multiphoton microscopy.
- p53 regulates renal expression of HIF-1{alpha} and pVHL under physiological conditions and after ischemia-reperfusion injury.
- Alteration of microvascular permeability in acute kidney injury.
- Impaired endothelial proliferation and mesenchymal transition contribute to vascular rarefaction following acute kidney injury.
- Fueling the fire in acute kidney injury: endothelial cells collect their Toll.
- MMP-9 gene deletion mitigates microvascular loss in a model of ischemic acute kidney injury.
- The p53 inhibitor pifithrin-α can stimulate fibrosis in a rat model of ischemic acute kidney injury.
- Soluble thrombomodulin reduces inflammation and prevents microalbuminuria induced
- Activated pericytes and the inhibition of renal vascular stability: obstacles for kidney repair.
Treatments
- Acute Kidney Failure, End-stage Renal Disease (esrd)
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