Dr. Richard P. Lifton M.D.
Internist
300 Cedar St Tac S341d New Haven CT, 06519About
Dr. Richard Lifton is an internist practicing in New Haven, CT. Dr. Lifton specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Lifton provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Education and Training
Stanford Univ Sch Of Med- Stanford Ca 1982
Stanford University School of Medicine 1982
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Paracellin-1, a renal tight junction protein required for paracellular Mg2+ resorption.
- Localization of a gene for autosomal recessive distal renal tubular acidosis with normal hearing (rdRTA2) to 7q33-34.
- Glucocorticoid-remediable aldosteronism.
- Activating mineralocorticoid receptor mutation in hypertension exacerbated by pregnancy.
- Mutations in ATP6N1B, encoding a new kidney vacuolar proton pump 116-kD subunit, cause recessive distal renal tubular acidosis with preserved hearing.
- Bartter syndrome and focal segmental glomerulosclerosis: a possible link between two diseases.
- Evidence for a gene influencing blood pressure on chromosome 17. Genome scan linkage results for longitudinal blood pressure phenotypes in subjects from the framingham heart study.
- IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
- Gitelman's syndrome revisited: an evaluation of symptoms and health-related quality of life.
- Molecular mechanisms of human hypertension.
- Mutations in the Na-Cl cotransporter reduce blood pressure in humans.
- Molecular cloning and characterization of Atp6n1b: a novel fourth murine vacuolar H+-ATPase a-subunit gene.
- Human hypertension caused by mutations in WNK kinases.
- High bone density due to a mutation in LDL-receptor-related protein 5.
- KRIT1, a gene mutated in cerebral cavernous malformation, encodes a microtubule-associated protein.
Clinical Trials
Dr. Richard P. Lifton M.D.'s Practice location
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