Dr. Howard J Goldberg M.D.
Gastroenterologist | Gastroenterology
12012 Veirs Mill Rd Silver Spring MD, 20906About
Dr. Howard Goldberg is a gastroenterologist practicing in Silver Spring, MD. Dr. Goldberg specializes in the digestive system and its diseases that affect the gastrointestinal tract, which include organs from the mouth to the anus as well as liver disorders. Gastroenterology includes conditions such as hepatitis, peptic ulcer disease, colitis, nutritional problems and irritable bowel syndrome. Dr. Goldberg performs colonoscopy and endoscopy procedures and provides accurate and thorough care for patients suffering from digestive issues.
Education and Training
Georgetown Univ Sch of Med, Washington Dc 1979
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mesangial cell filamentous actin disassembly and hypocontractility in high glucose are mediated by PKC-zeta.
- Overexpression of the type II transforming growth factor-beta receptor inhibits fibroblasts proliferation and activates extracellular signal regulated kinase and c-Jun N-terminal kinase.
- The hexosamine pathway regulates the plasminogen activator inhibitor-1 gene promoter and Sp1 transcriptional activation through protein kinase C-beta I and -delta.
- Take your Pill(Cam): it might save your life.
- In high glucose protein kinase C-zeta activation is required for mesangial cell generation of reactive oxygen species.
- Posttranslational, reversible O-glycosylation is stimulated by high glucose and mediates plasminogen activator inhibitor-1 gene expression and Sp1 transcriptional activity in glomerular mesangial cells.
- Reactive oxygen species, PKC-beta1, and PKC-zeta mediate high-glucose-induced vascular endothelial growth factor expression in mesangial cells.
- High glucose activates PKC-zeta and NADPH oxidase through autocrine TGF-beta1 signaling in mesangial cells.
- Rosiglitazone prevents high glucose-induced vascular endothelial growth factor and collagen IV expression in cultured mesangial cells.
- Inhibition of Src kinase blocks high glucose-induced EGFR transactivation and collagen synthesis in mesangial cells and prevents diabetic nephropathy in mice.
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