Dr. Elizabeth Deboran Mellins MD
Rheumatologist (Pediatric) | Pediatric Rheumatology
725 Welch Rd Palo Alto CA, 94304About
Dr. Elizabeth Mellins practices Pediatric Rheumatology in Palo Alto, CA. Pediatric rheumatologists treat child patients who suffer from pain in the musculoskeletal system, have symptoms of arthritis, or an autoimmune disorder. Dr. Mellins evaluates and treats a variety of joint, muscle, and bone disorders; including autoimmune disorders, such as lupus, juvenile rheumatoid arthritis, scleroderma, Kawasaki disease, post-infectious arthritis, chronic vasculitis, and inflammatory disorders of the muscle, eye, or other organs. Dr. Mellins also is trained in the evaluation of prolonged fever, unexplained complaints of chronic musculoskeletal pain, weakness, poor appetite, fatigue, and/or loss of function or skills.
Education and Training
Harvard Med Sch, Boston Ma 1978
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Accessory molecules for MHC class II peptide loading.
- Determination of the HLA-DM interaction site on HLA-DR molecules.
- Autoantigenic HCgp39 epitopes are presented by the HLA-DM-dependent presentation pathway in human B cells.
- Rheumatoid arthritis (RA)-associated HLA-DR alleles form less stable complexes with class II-associated invariant chain peptide than non-RA-associated HLA-DR alleles.
- Relationship between kinetic stability and immunogenicity of HLA-DR4/peptide complexes.
- Stabilization of soluble, low-affinity HLA-DM/HLA-DR1 complexes by leucine
- Labeling antigen-specific CD4(+) T cells with class II MHC oligomers.
- Latent cytomegalovirus down-regulates major histocompatibility complex class II expression on myeloid progenitors.
- Structural factors contributing to DM susceptibility of MHC class II/peptide complexes.
- Formation of two peptide/MHC II isomers is catalyzed differentially by HLA-DM.
- Point mutations in or near the antigen-binding groove of HLA-DR3 implicate class II-associated invariant chain peptide affinity as a constraint on MHC class II polymorphism.
- Susceptibility of immature and mature Langerhans cell-type dendritic cells to
- Cytokines elicited by T cell epitopes from a synovial autoantigen: altered peptide ligands can reduce interferon-gamma and interleukin-10 production.
- Regulation of the class II MHC pathway in primary human monocytes by granulocyte-macrophage colony-stimulating factor.
- Interaction of HLA-DR with an acidic face of HLA-DM disrupts sequence-dependent interactions with peptides.
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