Dr. Anthony J Infante MD
Allergist and Immunologist (Pediatric) | Pediatric Allergy/Immunology
333 N Santa Rosa Ave 8th Floor San Antonio TX, 78207About
Dr. Anthony Infante practices Pediatric Allergy Medicine in San Antonio, TX. A pediatric allergist/immunologist finds and treats allergies and immune system problems that child patients suffer from. Dr. Infante treats the reactions caused by pet dander, pollen, dust, mold spores, insect stings, food, and medications; those reactions include asthma, hay fever, hives, eczema, or a very severe and unusual reaction called anaphylaxis.
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Absence of a significant mixed lymphocyte reaction in a marsupial (Monodelphis domestica).
- Perspectives on T cell receptor expression in autoimmune disease.
- Myasthenia gravis and its animal model: T cell receptor expression in an antibody mediated autoimmune disease.
- TCR v(beta) repertoire restriction and lack of CDR3 conservation implicate TCR-superantigen interactions in promoting the clonal evolution of murine thymic lymphomas.
- Interleukin-12 enhances clinical experimental autoimmune myasthenia gravis in susceptible but not resistant mice.
- Chronic granulomatous disease and other disorders of neutrophil function.
- TCRBV CDR3 diversity of CD4+ and CD8+ T-lymphocytes in HIV-infected individuals.
- Cryptic determinants and promiscuous sequences on human acetylcholine receptor:
- Immunological memory and late onset autoimmunity.
- Split tolerance in a novel transgenic model of autoimmune myasthenia gravis.
- Evidence of a diverse T cell receptor repertoire for acetylcholine receptor, the autoantigen of myasthenia gravis.
- Recall immune memory: a new tool for generating late onset autoimmune myasthenia gravis.
- A hierarchy of T cell receptor motifs determines responsiveness to the immunodominant epitope in experimental autoimmune myasthenia gravis.
- Discrete T cell populations with specificity for a neo-self-antigen bear distinct imprints of tolerance.
- Maintenance of immune tolerance to a neo-self acetylcholine receptor antigen with aging: implications for late-onset autoimmunity.
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