Dr. Dwight Gary Gilliland MD
Hematologist-Oncologist | Hematology & Oncology
1 Blackfan Circle Room 5210 Childrens Boston MA, 02115About
Dr. Dwight Gilliland is a hematologist oncologist practicing in Boston, MA. Dr. Gilliland specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
Education and Training
University of California 1984
Provider Details
Expert Publications
Data provided by the National Library of Medicine- TEL/PDGFbetaR induces hematologic malignancies in mice that respond to a specific tyrosine kinase inhibitor.
- Transforming properties of the Huntingtin interacting protein 1/ platelet-derived growth factor beta receptor fusion protein.
- Haploinsufficiency of CBFA2 causes familial thrombocytopenia with propensity to develop acute myelogenous leukaemia.
- Fatal myeloproliferation, induced in mice by TEL/PDGFbetaR expression, depends on PDGFbetaR tyrosines 579/581.
- Signal transduction and transforming properties of the TEL-TRKC fusions associated with t(12;15)(p13;q25) in congenital fibrosarcoma and acute myelogenous leukemia.
- TEL/PDGFbetaR fusion protein activates STAT1 and STAT5: a common mechanism for transformation by tyrosine kinase fusion proteins.
- Divergent roles of STAT1 and STAT5 in malignancy as revealed by gene disruptions in mice.
- Stat5 is essential for the myelo- and lymphoproliferative disease induced by TEL/JAK2.
- Translocation (2;8)(p12;q24) associated with a cryptic t(12;21)(p13;q22) TEL/AML1 gene rearrangement in a child with acute lymphoblastic leukemia.
- Induction of myeloproliferative disease in mice by tyrosine kinase fusion oncogenes does not require granulocyte-macrophage colony-stimulating factor or interleukin-3.
- Interleukin 3 and granulocyte-macrophage colony-stimulating factor are not required for induction of chronic myeloid leukemia-like myeloproliferative disease in mice by BCR/ABL.
- ARG tyrosine kinase activity is inhibited by STI571.
- The diverse role of the ETS family of transcription factors in cancer.
- Socs-1 inhibits TEL-JAK2-mediated transformation of hematopoietic cells through
- Molecular genetics of acute myeloid leukaemia.
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