Dr. Reynold A Panettieri MD
Pulmonologist | Pulmonary Disease
51 N 39th St Phi 1st Floor Rear Philadelphia PA, 19104About
Dr. Reynold Panettieri practices Pulmonology in Philadelphia, PA. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Panettieri manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- alpha-Thrombin stimulates contraction of human bronchial rings by activation of protease-activated receptors.
- Phosphatidylinositol 3-kinase mediates mitogen-induced human airway smooth muscle cell proliferation.
- Regulation of gelatinases in human airway smooth muscle cells: mechanism of progelatinase A activation.
- Up-regulation of ICAM-1 by cytokines in human tracheal smooth muscle cells involves an NF-kappa B-dependent signaling pathway that is only partially sensitive to dexamethasone.
- A comprehensive educational program improves clinical outcome measures in inner-city patients with asthma.
- MAPK superfamily activation in human airway smooth muscle: mitogenesis requires prolonged p42/p44 activation.
- Inflammatory and contractile agents sensitize specific adenylyl cyclase isoforms in human airway smooth muscle.
- Glucocorticoids ablate IL-1beta-induced beta-adrenergic hyporesponsiveness in human airway smooth muscle cells.
- Role of ERK MAP kinases in responses of cultured human airway smooth muscle cells to IL-1beta.
- Activation of K-p21ras and N-p21ras, but not H-p21ras, is necessary for mitogen-induced human airway smooth-muscle proliferation.
- Activation of p55 tumor necrosis factor-alpha receptor-1 coupled to tumor necrosis factor receptor-associated factor 2 stimulates intercellular adhesion molecule-1 expression by modulating a thapsigargin-sensitive pathway in human tracheal smooth mus
- Mechanisms of proliferation synergy by receptor tyrosine kinase and G protein-coupled receptor activation in human airway smooth muscle.
- p38 MAP kinase regulates IL-1 beta responses in cultured airway smooth muscle cells.
- Tumor necrosis factor-alpha-induced secretion of RANTES and interleukin-6 from human airway smooth-muscle cells. Modulation by cyclic adenosine monophosphate.
- Polymorphism of the beta(2)-adrenergic receptor gene and desensitization in human airway smooth muscle.
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