Dr. Craig Alan Henke M.D.
Pulmonologist | Pulmonary Disease
University Of Minnesota Physicians 516 Delaware Street Minneapolis MN, 55455About
Dr. Craig Henke practices Pulmonology in Minneapolis, MN. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Henke manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
Univ of Mn Med Sch-Minneapolis, Minneapolis Mn 1983
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Critical Care Medicine
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Induction of lung fibroblast apoptosis by soluble fibronectin peptides.
- Focal adhesion kinase is upstream of phosphatidylinositol 3-kinase/Akt in regulating fibroblast survival in response to contraction of type I collagen matrices via a beta 1 integrin viability signaling pathway.
- Profound hypoxemia precipitated by positive end-expiratory pressure: induction of an intracardiac shunt.
- Role of integrin-linked kinase in regulating phosphorylation of Akt and fibroblast survival in type I collagen matrices through a beta1 integrin viability signaling pathway.
- Lung transplant pathology. A comparative study of pulmonary acute rejection and cytomegaloviral infection.
- PTEN regulates fibroblast elimination during collagen matrix contraction.
- Polymerized collagen inhibits fibroblast proliferation via a mechanism involving the formation of a beta1 integrin-protein phosphatase 2A-tuberous sclerosis complex 2 complex that suppresses S6K1 activity.
- Fibrotic myofibroblasts manifest genome-wide derangements of translational control.
- Fibroproliferative disorders.
- Pathologic caveolin-1 regulation of PTEN in idiopathic pulmonary fibrosis.
- Telomerase and telomere length in pulmonary fibrosis.
- FoxO3a (Forkhead Box O3a) deficiency protects Idiopathic Pulmonary Fibrosis (IPF) fibroblasts from type I polymerized collagen matrix-induced apoptosis via caveolin-1 (cav-1) and Fas.
- Identification of a cell-of-origin for fibroblasts comprising the fibrotic reticulum in idiopathic pulmonary fibrosis.
- miR-210 promotes IPF fibroblast proliferation in response to hypoxia.
- Pathologic Regulation of Collagen I by an Aberrant Protein Phosphatase 2A/Histone Deacetylase C4/MicroRNA-29 Signal Axis in Idiopathic Pulmonary Fibrosis Fibroblasts.
Fellowships
- University of Minnesota Hospital
- University of Minnesota Hospital
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