Dr. William Scott Griffin D.D.S.
Dentist | General Practice
12520 CAPITAL BLVD WAKE FOREST NC, 27587About
Dr. William Griffin is a Dentist practicing in WAKE FOREST, NC. Dr. Griffin specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Is there a genetic basis for the deposition of beta-amyloid after fatal head injury?
- Alterations in synaptic proteins and their encoding mRNAs in prefrontal cortex in schizophrenia: a possible neurochemical basis for 'hypofrontality'.
- Increased interleukin-1beta converting enzyme expression and activity in Alzheimer disease.
- Evidence for caspase-mediated cleavage of AMPA receptor subunits in neuronal apoptosis and Alzheimer's disease.
- Mitogen-activated protein kinases in schizophrenia.
- Overexpression of s100beta in Down's syndrome: correlation with patient age and with beta-amyloid deposition.
- S100beta induction of the proinflammatory cytokine interleukin-6 in neurons.
- Overexpression of the neuritotrophic cytokine S100beta precedes the appearance of neuritic beta-amyloid plaques in APPV717F mice.
- Neuronal-glial interactions mediated by interleukin-1 enhance neuronal acetylcholinesterase activity and mRNA expression.
- Association of early-onset Alzheimer's disease with an interleukin-1alpha gene
- Association of interleukin-1 gene polymorphisms with Alzheimer's disease.
- Interleukin-1 promotes expression and phosphorylation of neurofilament and tau proteins in vivo.
- Interleukin-1 and the immunogenetics of Alzheimer disease.
- Phospholipid abnormalities in postmortem schizophrenic brains detected by 31P nuclear magnetic resonance spectroscopy: a preliminary study.
- Interleukin-1 promotion of MAPK-p38 overexpression in experimental animals and in Alzheimer's disease: potential significance for tau protein phosphorylation.
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