Dr. Jonathan Harry Schatz M.D.
Oncologist | Medical Oncology
401 E 89TH ST APT 7C NEW YORK NY, 10128About
Dr. Jonathan Schatz is an oncologist practicing in NEW YORK, NY. Dr. Schatz specializes in the care and treatment of patients with cancer. As an oncologist, Dr. Schatz manages and oversees the treatment of a cancer patient after he or she has been diagnosed with the disease. Oncologists will care for their patients throughout the course of the disease. Types of oncologists include medical oncologists, surgical oncologists, radiation oncologists, gynecologic oncologists, pediatric oncologists and hematologist oncologists.
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Expert Publications
Data provided by the National Library of Medicine- Graded expression of interferon regulatory factor-4 coordinates isotype switching with plasma cell differentiation.
- Sirolimus (rapamycin) induced proteinuria in a patient undergoing allogeneic hematopoietic stem cell transplant.
- Cooperation between deficiencies of IRF-4 and IRF-8 promotes both myeloid and lymphoid tumorigenesis.
- Mouse models of cancer as biological filters for complex genomic data.
- Targeting the PI3K/AKT/mTOR pathway in non-Hodgkin's lymphoma: results, biology, and development strategies.
- The Eph-receptor A7 is a soluble tumor suppressor for follicular lymphoma.
- Targeted cancer therapy: what if the driver is just a messenger?
- Progress against follicular lymphoma.
- Frequent disruption of the RB pathway in indolent follicular lymphoma suggests a new combination therapy.
- RNA G-quadruplexes cause eIF4A-dependent oncogene translation in cancer.
- A rare presentation of in situ mantle cell lymphoma and follicular lymphoma: a case report and review of the literature.
- Synergistic induction of apoptosis in high-risk DLBCL by BCL2 inhibition with ABT-199 combined with pharmacologic loss of MCL1.
- Evidence Suggesting That Discontinuous Dosing of ALK Kinase Inhibitors May Prolong Control of ALK+ Tumors.
- Kinase overexpressing cancers responsive to drug withdrawal.
- Oncogene Overdose: Too Much of a Bad Thing for Oncogene-Addicted Cancer Cells.
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