Dr. Matthew Thomas Rondina M.D.
Internist
Va Slc Hcs 500 Foothill Drive Salt Lake City UT, 84148About
Dr. Matthew Rondina is an internist practicing in Salt Lake City, UT. Dr. Rondina specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Rondina provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The effects of drilling force on cortical temperatures and their duration: an in vitro study.
- Abdominal aortitis due to Streptococcus pneumoniae and Enterobacter aerogenes: a case report and review.
- Azathioprine-induced warfarin resistance.
- Contemporary issues in the prevention and management of postthrombotic syndrome.
- Peri-procedural anticoagulation in patients undergoing ablation for atrial fibrillation.
- Drug-induced thrombocytopenia for the hospitalist physician with a focus on heparin-induced thrombocytopenia.
- Platelet-leukocyte interactions link inflammatory and thromboembolic events in ischemic stroke.
- Prevention of venous thromboembolism in obesity.
- ACP Journal Club. Review: Graduated compression stockings reduce deep venous thrombosis in hospitalized patients.
- Prospective comparison of three enoxaparin dosing regimens to achieve target anti-factor Xa levels in hospitalized, medically ill patients with extreme obesity.
- Targeting phosphodiesterases in anti-platelet therapy.
- Mammalian target of rapamycin regulates neutrophil extracellular trap formation via induction of hypoxia-inducible factor 1 α.
- Argatroban dose reductions for suspected heparin-induced thrombocytopenia complicated by child-pugh class C liver disease.
- A sticky story for signal transducer and activator of transcription 3 in platelets.
- Methicillin-resistant Staphylococcus aureus-induced thrombo-inflammatory response is reduced with timely antibiotic administration.
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