Dr. Lloyd B. Klickstein MD, PHD
Rheumatologist | Rheumatology
400 Technology Sq Novartis Institute F Cambridge MA, 02139About
Dr. Lloyd Klickstein is a rheumatologist practicing in Cambridge, MA. Dr. Klickstein specializes in the treatment of musculoskeletal diseases and systematic autoimmune conditions that can affect the bones, muscles or bones. Eventually, if not treated, these illnesses can also impact the skin, eyes, nervous system and internal organs. Dr. Klickstein treats diseases similar to orthopedists but does not perform surgery. Often times, research is conducted to find potential alternatives for the patients illness.
Education and Training
Harvard Medical School 1989
Provider Details
Expert Publications
Data provided by the National Library of Medicine- C1q-binding proteins and C1q receptors.
- Specific activation of leukocyte beta2 integrins lymphocyte function-associated antigen-1 and Mac-1 by chemokines mediated by distinct pathways via the alpha subunit cytoplasmic domains.
- C1q and C4b bind simultaneously to CR1 and additively support erythrocyte adhesion.
- Intercellular adhesion molecule 1 and beta2 integrins in C1q-stimulated superoxide production by human neutrophils: an example of a general regulatory mechanism governing acute inflammation.
- C5a-stimulated human neutrophils use a subset of beta2 integrins to support the adhesion-dependent phase of superoxide production.
- Complement receptor 1/CD35 is a receptor for mannan-binding lectin.
- Dual role of H-Ras in regulation of lymphocyte function antigen-1 activity by stromal cell-derived factor-1alpha: implications for leukocyte transmigration.
- Lack of age-associated LFA-1 up-regulation and impaired ICAM-1 binding in lymphocytes from patients with Down syndrome.
- A dominant Jurkat T cell mutation that inhibits LFA-1-mediated cell adhesion is associated with increased cell growth.
- A comparison of type I collagen, fibronectin, and vitronectin in supporting adhesion of mechanically strained osteoblasts.
- Expression and function of C1q receptors and C1q binding proteins at the cell surface.
- Calreticulin is at the surface of circulating neutrophils and uses CD59 as an adaptor molecule.
- A role for lipid rafts in C1q-triggered O2- generation by human neutrophils.
- RhoH is required to maintain the integrin LFA-1 in a nonadhesive state on lymphocytes.
- The integrin alpha-subunit leg extends at a Ca2+-dependent epitope in the thigh/genu interface upon activation.
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