Dr. Alexei A Grom M.D.
Rheumatologist (Pediatric) | Pediatric Rheumatology
3333 Burnet Ave Ml 4010 Cincinnati OH, 45229About
Dr. Alexei Grom practices Pediatric Rheumatology in Cincinnati, OH. Pediatric rheumatologists treat child patients who suffer from pain in the musculoskeletal system, have symptoms of arthritis, or an autoimmune disorder. Dr. Grom evaluates and treats a variety of joint, muscle, and bone disorders; including autoimmune disorders, such as lupus, juvenile rheumatoid arthritis, scleroderma, Kawasaki disease, post-infectious arthritis, chronic vasculitis, and inflammatory disorders of the muscle, eye, or other organs. Dr. Grom also is trained in the evaluation of prolonged fever, unexplained complaints of chronic musculoskeletal pain, weakness, poor appetite, fatigue, and/or loss of function or skills.
Education and Training
Leningrad Pediatric Med Inst, Leningrad, Russia 1986
St. Petersburg State Pediatric-Medical University 1985
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- T-cell and T-cell receptor abnormalities in the immunopathogenesis of juvenile rheumatoid arthritis.
- Expression of angiogenic factors in juvenile rheumatoid arthritis: correlation
- Etanercept in the treatment of macrophage activation syndrome.
- Interleukin-15 inhibits sodium nitroprusside-induced apoptosis of synovial fibroblasts and vascular endothelial cells.
- Macrophage activation syndrome and reactive hemophagocytic lymphohistiocytosis: the same entities?
- Natural killer cell dysfunction: A common pathway in systemic-onset juvenile rheumatoid arthritis, macrophage activation syndrome, and hemophagocytic lymphohistiocytosis?
- Gene expression in juvenile arthritis and spondyloarthropathy: pro-angiogenic ELR+ chemokine genes relate to course of arthritis.
- Natural killer cell dysfunction is a distinguishing feature of systemic onset juvenile rheumatoid arthritis and macrophage activation syndrome.
- Does systemic-onset juvenile idiopathic arthritis belong under juvenile idiopathic arthritis?
- Association between lack of angiogenic response in muscle tissue and high expression of angiostatic ELR-negative CXC chemokines in patients with juvenile dermatomyositis: possible link to vasculopathy.
- The diagnostic significance of soluble CD163 and soluble interleukin-2 receptor alpha-chain in macrophage activation syndrome and untreated new-onset systemic juvenile idiopathic arthritis.
- B cell depletion: on the rise.
- Gene expression profiling of peripheral blood from patients with untreated new-onset systemic juvenile idiopathic arthritis reveals molecular heterogeneity that may predict macrophage activation syndrome.
- Macrophage activation syndrome in patients with systemic juvenile idiopathic arthritis is associated with MUNC13-4 polymorphisms.
- Systemic JIA: new developments in the understanding of the pathophysiology and therapy.
Treatments
- Psoriasis
- Arthritis
- Lupus
- Pain
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