Samuel Albert Camacho M.D.
Cardiologist | Cardiovascular Disease
10201 Se Main St Suite 10 Portland OR, 97216About
Dr. Samuel Camacho is a cardiologist practicing in Portland, OR. Dr. Camacho specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Camacho also practices preventative medicine, helping patients maintain a heart-healthy life.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Role of slowed Ca(2+) transient decline in slowed relaxation during myocardial ischemia.
- Chronic dipyridamole therapy produces sustained protection against cardiac ischemia-reperfusion injury.
- CXCR5-deficient mice develop functional germinal centers in the splenic T cell zone.
- Affinity maturation in ectopic germinal centers.
- Preconditioning limits mitochondrial Ca(2+) during ischemia in rat hearts: role of K(ATP) channels.
- Vaccination with glutamic acid decarboxylase plasmid DNA protects mice from
- A key role for ICAM-1 in generating effector cells mediating inflammatory responses.
- Suppression of motion artifacts in fluorescence spectroscopy of perfused hearts.
- Cardiac contractile dysfunction during mild coronary flow reductions is due to an altered calcium-pressure relationship in rat hearts.
- Relationship between myocardial metabolites and contractile abnormalities during graded regional ischemia. Phosphorus-31 nuclear magnetic resonance studies of porcine myocardium in vivo.
- The effect of dobutamine on myocardial performance and high-energy phosphate metabolism at different stages of heart failure in cardiomyopathic hamsters: a 31P MRS study.
- Immunoassay quantification using surface-enhanced fluorescence (SEF) tags.
- 31P and 1H magnetic resonance spectroscopy of acute alcohol cardiac depression in rats.
- Basic mechanisms of myocardial dysfunction: cellular pathophysiology of heart failure.
- Protein and acidosis alter calcium-binding and fluorescence spectra of the calcium indicator indo-1.
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