Matthew Ryan Prater M.D.
Internist
1224 TROTWOOD AVE COLUMBIA TN, 38401About
Dr. Matthew Prater is an internist practicing in Jackson, MS. Dr. Prater specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Prater provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Education and Training
University of Mississippi School of Medicine 2012
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Characterization of metastatic intestinal adenocarcinoma with differentiation into multiple morphologic cell types in a Virginia opossum.
- Diffuse annular fusiform adenocarcinoma in a dog.
- Bilateral dissected spleens and thymuses in rodents exhibit homogeneity in leukocyte markers.
- Maternal immune stimulation in mice decreases fetal malformations caused by teratogens.
- Single-dose topical exposure to the pyrethroid insecticide, permethrin in C57BL/6N mice: effects on thymus and spleen.
- Cis-urocanic acid increases immunotoxicity and lethality of dermally administered permethrin in C57BL/6N mice.
- Indirect effect of guanine nucleotides on antagonist binding to A1 adenosine receptors: occupation of cryptic binding sites by endogenous vesicular adenosine.
- Molecular mechanisms of cis-urocanic acid and permethrin-induced alterations in cutaneous immunity.
- Combined dermal exposure to permethrin and cis-urocanic acid suppresses the contact hypersensitivity response in C57BL/6N mice in an additive manner.
- Does immune stimulation or antioxidant therapy reduce MNU-induced placental damage via activation of Jak-STAT and NFkappaB signaling pathways?
- Contamination of adenosine deaminase by superoxide dismutase. Stabilization of endothelium-derived relaxing factor.
- Placental oxidative stress alters expression of murine osteogenic genes and impairs fetal skeletal formation.
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