Dr. William David Bloomer M.D.
Doctor
2650 Ridge Ave Evanston IL, 60201About
Dr. William David Bloomer M.D. is a top Doctor in Evanston, IL. With a passion for the field and an unwavering commitment to their specialty, Dr. William David Bloomer M.D. is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Dr. William David Bloomer M.D. is a prime example of a true leader in health care. As a leader and expert in their field, Dr. William David Bloomer M.D. is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In Evanston, IL, Dr. William David Bloomer M.D. is a true asset to their field and dedicated to the profession of medicine.
Education and Training
Jefferson Med Coll-Thos Jefferson Univ, Philadelphia Pa 1970
Sidney Kimmel Medical College at Thomas Jefferson University 1970
Board Certification
Nuclear MedicineAmerican Board of Nuclear MedicineABNM
RadiologyAmerican Board of RadiologyABR
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mechanisms involved in the potentiation of melphalan by the bioreductive compound THNLA-1 in vitro.
- Normal tissue responses to radiation therapy.
- Lumpectomy and breast irradiation for breast cancer arising after previous radiotherapy for Hodgkin's disease or lymphoma.
- 4-[3-(2-Nitro-1-imidazolyl)propylamino]-7-chloroquinoline hydrochloride (NLCQ-1),
- Schedule-dependent potentiation of chemotherapeutic drugs by the bioreductive compounds NLCQ-1 and tirapazamine against EMT6 tumors in mice.
- 4-[3-(2-Nitro-1-imidazolyl)propylamino]-7-chloroquinoline hydrochloride (NLCQ-1), a novel bioreductive agent as radiosensitizer in vitro and in vivo: comparison with tirapazamine.
- In vitro evaluation of 4-[3-(2-nitro-1-imidazolyl)-propylamino]-7-trifluoromethylquinoline hydrochloride (NLTQ-1), a new bioreductive agent as a hypoxia marker by 19F-magnetic resonance spectroscopy (19F-MRS).
- Synergistic enhancement of the antitumor effect of taxol by the bioreductive compound NLCQ-1, in vivo: comparison with tirapazamine.
- Therapeutic advantage from combining 5-fluorouracil with the hypoxia-selective cytotoxin NLCQ-1 in vivo; comparison with tirapazamine.
- Enhancement of the antitumor effect of cyclophosphamide with the hypoxia-selective cytotoxin NLCQ-1 against murine tumors and human xenografts.
- Therapeutic advantage from combining paclitaxel with the hypoxia-selective cytotoxin NLCQ-1 in murine tumor- or human xenograft-bearing mice.
- Synergistic interaction between cyclophosphamide or paclitaxel and the bioreductive compound NLCPQ-1, in vivo.
- Potentiation of 1-(2-chloroethyl)-3-cyclohexyl-1-nitrosourea's toxicity in vitro by two new bioreductive agents.
- Reductive metabolism of the nitroimidazole-based hypoxia-selective cytotoxin NLCQ-1 (NSC 709257).
- NLCQ-1 (NSC 709257): exploiting hypoxia with a weak DNA-intercalating bioreductive drug.
Treatments
- Breast Cancer
- Prostate Cancer
- Lymphoma
- Pancreatic Cancer
- Brain Tumor
- Lung Cancer
- Esophageal Cancer
- Aerolase® Technologies
- Extra Corporeal Shockwave Therapy
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