Dr. Barbara I Kazmierczak MD
Infectious Disease Specialist | Infectious Disease
789 Howard Ave Dana Building 3rd Fl New Haven CT, 06519About
Dr. Barbara Kazmierczak is an infectious disease specialist practicing in New Haven, CT. Dr. Kazmierczak specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
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Expert Publications
Data provided by the National Library of Medicine- A novel sensor kinase-response regulator hybrid regulates type III secretion and is required for virulence in Pseudomonas aeruginosa.
- An indirect enzyme-linked immunosorbent assay for rapid and quantitative assessment of Type III virulence phenotypes of Pseudomonas aeruginosa isolates.
- Analysis of FimX, a phosphodiesterase that governs twitching motility in
- Mutational analysis of RetS, an unusual sensor kinase-response regulator hybrid required for Pseudomonas aeruginosa virulence.
- FlhF is required for swimming and swarming in Pseudomonas aeruginosa.
- Pseudomonas aeruginosa chronic colonization in cystic fibrosis patients.
- Pseudomonas aeruginosa exhibits sliding motility in the absence of type IV pili and flagella.
- Immune recognition of Pseudomonas aeruginosa mediated by the IPAF/NLRC4 inflammasome.
- Pseudomonas aeruginosa OspR is an oxidative stress sensing regulator that affects pigment production, antibiotic resistance and dissemination during infection.
- Swarming motility, secretion of type 3 effectors and biofilm formation phenotypes exhibited within a large cohort of Pseudomonas aeruginosa clinical isolates.
- In vivo discrimination of type 3 secretion system-positive and -negative Pseudomonas aeruginosa via a caspase-1-dependent pathway.
- A biosynthetic strategy for re-engineering the Staphylococcus aureus cell wall with non-native small molecules.
- Airway epithelial MyD88 restores control of Pseudomonas aeruginosa murine infection via an IL-1-dependent pathway.
- Innate immune responses to Pseudomonas aeruginosa infection.
- The carbon monoxide releasing molecule CORM-2 attenuates Pseudomonas aeruginosa biofilm formation.
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