Dr. William Fleet Jackson DDS
Dentist | General Practice
509 W 15th St Washington NC, 27889About
Dr. William Jackson is a Dentist practicing in Washington, NC. Dr. Jackson specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
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Expert Publications
Data provided by the National Library of Medicine- CB(1) receptor antagonist SR141716A inhibits Ca(2+)-induced relaxation in CB(1) receptor-deficient mice.
- Hypoxia inhibits contraction but not calcium channel currents or changes in intracellular calcium in arteriolar muscle cells.
- K+-induced dilation of hamster cremasteric arterioles involves both the Na+/K+-ATPase and inward-rectifier K+ channels.
- Special edition of Microcirculation commemorating the 50th anniversary of the Microcirculatory Society, Inc.
- Potassium channels in the peripheral microcirculation.
- Membrane hyperpolarization is not required for sustained muscarinic agonist-induced increases in intracellular Ca2+ in arteriolar endothelial cells.
- Potassium channels and proliferation of vascular smooth muscle cells.
- Arteriolar smooth muscle Ca2+ dynamics during blood flow control in hamster cheek pouch.
- Inward rectifying potassium channels facilitate cell-to-cell communication in hamster retractor muscle feed arteries.
- Silent inward rectifier K+ channels in hypercholesterolemia.
- Vascular smooth muscle store-operated Ca2+ channels: what a TRP!
- Activation of potassium channels by tempol in arterial smooth muscle cells from normotensive and deoxycorticosterone acetate-salt hypertensive rats.
- Vanishing act: protein kinase C-dependent internalization of adenosine 5'-triphosphate-sensitive K+ channels.
- Connexin isoform expression in smooth muscle cells and endothelial cells of hamster cheek pouch arterioles and retractor feed arteries.
- KV1.3: a new therapeutic target to control vascular smooth muscle cell proliferation.
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