Dr. Robert O Heuckeroth MD
Gastroenterologist (Pediatric) | Pediatric Gastroenterology
1 Childrens Pl Saint Louis MO, 63110About
Dr. Robert Heuckeroth is a pediatric gastroenterologist practicing in Saint Louis, MO. Dr. Heuckeroth specializes in digestive system, liver or nutritional problems in children from the newborn period to their teens. Conditions such as irritable bowel syndrome, lactose intolerance, chronic constipation or obesity can all be treated by a pediatric gastroenterologist.
Education and Training
Washington Univ Sch Of Med- St Louis Mo 1990
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Anomalous development of the hepatobiliary system in the Inv mouse.
- RET signaling is essential for migration, axonal growth and axon guidance of developing sympathetic neurons.
- GDNF availability determines enteric neuron number by controlling precursor proliferation.
- Finding your way to the end: a tale of GDNF and endothelin-3.
- Neurturin signalling via GFRalpha2 is essential for innervation of glandular but not muscle targets of sacral parasympathetic ganglion neurons.
- GFRalpha1 expression in cells lacking RET is dispensable for organogenesis and nerve regeneration.
- A human yeast artificial chromosome containing the multiple endocrine neoplasia type 2B Ret mutation does not induce medullary thyroid carcinoma but does support the growth of kidneys and partially rescues enteric nervous system development in Ret-de
- Enteric neuroblasts require the phosphatidylinositol 3-kinase/Akt/Forkhead pathway for GDNF-stimulated survival.
- Transcriptional profiling after bile duct ligation identifies PAI-1 as a contributor to cholestatic injury in mice.
- Getting to the guts of enteric nervous system development.
- Differential gene expression and functional analysis implicate novel mechanisms in enteric nervous system precursor migration and neuritogenesis.
- BMP signaling regulates murine enteric nervous system precursor migration, neurite fasciculation, and patterning via altered Ncam1 polysialic acid addition.
- Reduced endothelin converting enzyme-1 and endothelin-3 mRNA in the developing bowel of male mice may increase expressivity and penetrance of Hirschsprung disease-like distal intestinal aganglionosis.
- Tissue-type plasminogen activator deficiency exacerbates cholestatic liver injury in mice.
- PAI-1 deficiency reduces liver fibrosis after bile duct ligation in mice through activation of tPA.
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PHILADELPHIA, PA 19104Get Direction
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