Dr. Clay F Semenkovich MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
4921 Parkview Pl Suite 13b Saint Louis MO, 63110About
Dr. Clay Semenkovich practices Endocrinology in Saint Louis, MO. Dr. Semenkovich specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Semenkovich examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Washington Univ Sch Of Med- St Louis Mo 1981
Washington Center / School of Medicine 1981
Washington University in St. Louis School of Medicine 1981
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Genetics and molecular biology.
- Respiratory uncoupling induces delta-aminolevulinate synthase expression through a nuclear respiratory factor-1-dependent mechanism in HeLa cells.
- Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in vivo.
- Relative hypoglycemia and hyperinsulinemia in mice with heterozygous lipoprotein lipase (LPL) deficiency. Islet LPL regulates insulin secretion.
- The role of osteoprogenitors in vascular calcification.
- Macrophage lipoprotein lipase promotes foam cell formation and atherosclerosis in low density lipoprotein receptor-deficient mice.
- Exercise induces lipoprotein lipase and GLUT-4 protein in muscle independent of adrenergic-receptor signaling.
- Skeletal muscle respiratory uncoupling prevents diet-induced obesity and insulin resistance in mice.
- Glucose and insulin stimulate heparin-releasable lipoprotein lipase activity in mouse islets and INS-1 cells. A potential link between insulin resistance and beta-cell dysfunction.
- Resistance exercise decreases skeletal muscle tumor necrosis factor alpha in frail elderly humans.
- PPARalpha deficiency reduces insulin resistance and atherosclerosis in apoE-null mice.
- The pancreatic beta cell heats up: UCP2 and insulin secretion in diabetes.
- Leucine and insulin activate p70 S6 kinase through different pathways in human skeletal muscle.
- PPARalpha suppresses insulin secretion and induces UCP2 in insulinoma cells.
- Respiratory uncoupling lowers blood pressure through a leptin-dependent mechanism in genetically obese mice.
Treatments
- Diabetes
- High Cholesterol
- Osteoporosis
- Type 2 Diabetes
- Hyperthyroidism
- Hypothyroidism
- Vascular Disease
- Goiter
- Graves' Disease
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