Therese Elaine Lane MS., MED
Speech-Language Pathologist
48 DENVER RD KINGSTON NY, 12401About
Dr. Therese Lane is a speech language pathologist practicing in KINGSTON, NY. Dr. Lane specializes in speech, language and swallowing disorders in patients. As a speech language pathologist, Dr. Lane evaluates, diagnoses and treats patients with communication and swallowing troubles. These conditions may be due to developmental delay, brain injury, hearing loss, autism, stroke or other diseases and injuries. Dr. Lane helps patients make sounds and improve their voices through various methods. Speech language pathologists also work with patients to strengthen muscles used to speak and swallow, and work with individuals and families to help cope with their conditions.
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Expert Publications
Data provided by the National Library of Medicine- Inhibition of nitric oxide synthase-2 reduces the severity of mouse hepatitis virus-induced demyelination: implications for NOS2/NO regulation of chemokine expression and inflammation.
- Protective immunity against murine hepatitis virus (MHV) induced by intranasal or subcutaneous administration of hybrids of tobacco mosaic virus that carries an MHV epitope.
- Viral-induced neurodegenerative disease.
- T cell attractant chemokine expression initiates lacrimal gland destruction in nonobese diabetic mice.
- The T cell chemoattractant IFN-inducible protein 10 is essential in host defense against viral-induced neurologic disease.
- Expression of Mig (monokine induced by interferon-gamma) is important in T lymphocyte recruitment and host defense following viral infection of the central nervous system.
- Regional hypomyelination and dysplasia in transgenic mice with astrocyte-directed expression of interferon-gamma.
- Reduced macrophage infiltration and demyelination in mice lacking the chemokine receptor CCR5 following infection with a neurotropic coronavirus.
- Neutralization of the chemokine CXCL10 reduces inflammatory cell invasion and demyelination and improves neurological function in a viral model of multiple sclerosis.
- Lack of CCR2 results in increased mortality and impaired leukocyte activation and trafficking following infection of the central nervous system with a neurotropic coronavirus.
- Chemokine expression and viral infection of the central nervous system: regulation of host defense and neuropathology.
- The CXC chemokines IP-10 and Mig are essential in host defense following infection with a neurotropic coronavirus.
- Functional diversity of chemokines and chemokine receptors in response to viral infection of the central nervous system.
- Human embryonic stem cell-derived oligodendrocyte progenitor cells express the serotonin receptor and are susceptible to JC virus infection.
- Iron limitation and the gamma interferon-mediated antihistoplasma state of murine macrophages.
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