Dr. Paul J Christensen MD
Pulmonologist | Pulmonary Disease
1500 East Medical Center Dr 3rd Floor Taubman Ct Ann Arbor MI, 48109About
Dr. Paul Christensen practices Pulmonology in Ann Arbor, MI. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Christensen manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
Univ of Mn Med Sch-Minneapolis, Minneapolis Mn 1985
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Alveolar epithelial cell inhibition of fibroblast proliferation is regulated by MCP-1/CCR2 and mediated by PGE2.
- Transgenic overexpression of granulocyte macrophage-colony stimulating factor in the lung prevents hyperoxic lung injury.
- Expression and functional implications of CCR2 expression on murine alveolar epithelial cells.
- Quantitative exposure assessment for confinement of maize biogenic systems.
- Expression of the reverse tetracycline-transactivator gene causes emphysema-like changes in mice.
- GM-CSF and the impaired pulmonary innate immune response following hyperoxic stress.
- Disparate mechanisms of sICAM-1 production in the peripheral lung: contrast between alveolar epithelial cells and pulmonary microvascular endothelial cells.
- Co-infection with pansensitive and multidrug-resistant strains of Mycobacterium tuberculosis.
- Curcumin inhibits fibrosis-related effects in IPF fibroblasts and in mice following bleomycin-induced lung injury.
- The vitamin D/CYP24A1 story in cancer.
- Overexpression of sICAM-1 in the alveolar epithelial space results in an exaggerated inflammatory response and early death in Gram negative pneumonia.
- Vitamin D and lung cancer.
- Repeated exposure to Aspergillus fumigatus conidia results in CD4+ T cell-dependent and -independent pulmonary arterial remodeling in a mixed Th1/Th2/Th17 microenvironment that requires interleukin-4 (IL-4) and IL-10.
- Advanced intimal hyperplasia without luminal narrowing of leptomeningeal arteries in CADASIL.
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