Dr. William Judson Burtis M.D.
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
747 Main St Suite 123 Concord MA, 01742About
Dr. William Burtis practices Endocrinology in Concord, MA. Dr. Burtis specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Burtis examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology and Metabolism
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Preliminary characterization of circulating amino- and carboxy-terminal fragments of parathyroid hormone-related peptide in humoral hypercalcemia of malignancy.
- Parathyroid hormone-related protein: structure, function, and measurement.
- Increased plasma protein binding and lower metabolic clearance rate of aldosterone in plasma of low cortisol concentration.
- Increased plasma protein binding and lower metabolic clearance rate of aldosterone in plasma of low cortisol concentration.
- Immunoaffinity purification of parathyroid hormone-related protein from bovine
- Measurement of circulating parathyroid hormone-related protein in rats with humoral hypercalcemia of malignancy using a two-site immunoradiometric assay.
- Humoral hypercalcemia of cancer. Identification of a novel parathyroid hormone-like peptide.
- Co-purification of transforming growth factor beta-like activity with PTH-like and bone-resorbing activities from a tumor associated with humoral hypercalcemia of malignancy.
- Hypocalcemia associated with calcium-soap formation in a patient with a pancreatic fistula.
- Chemical abnormalities.
- Parathyroid hormone-related peptide and 8701-BC breast cancer cell growth and invasion in vitro: evidence for growth-inhibiting and invasion-promoting effects.
- Relative overexpression of the parathyroid hormone-related protein gene in human leiomyomas.
- Accumulation of carboxy-terminal fragments of parathyroid hormone-related protein
- Overexpression of parathyroid hormone-related protein in the pancreatic islets of transgenic mice causes islet hyperplasia, hyperinsulinemia, and hypoglycemia.
- Clonal variation of p53 expression and proliferative phenotype in A253 squamous carcinoma cells.
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